I recently had some correspondence with a company who have designed a device which supposedly mitigates EMF to some extent.
At first, I thought "just another company trying to sell some product which claims to block EMF". However, I was intrigued to learn that this device does NOT claim to block EMF. In fact, the mechanism appears to approach this problem from an entirely different angle, and so seems to be quite unique.
In short - It claims to be acting via resonance to maintain the binding of calcium ions within the proteinacous cavity, thereby modulating the VGCC threshold and preventing calcium efflux caused by external EMFs.
It has support from some of the big names in water science, including Professor Marc Henry.
For those who are not familiar with Professor Martin Pall's work + here(HIGHLY recommended), he has published several papers on the mechanisms of EMF toxicity. External EMF sources seem to activate voltage gated calcium channels inside the cell, which cause excessive calcium flooding (which has numerous downstream effects including mitochondrial damage, apoptosis, excess nitric oxide synthesis etc). Likewise, it has been proposed that these EMFs are potentially altering the biophysical properties of the calcium ion via something called cyclotron resonance (if I understand correctly):
Interestingly, many of the biological effects of EMF can be mitigated via calcium channel blockers, which strongly implicates VGCC activation being one of the primary mechanisms of cellular stress. The CMO supposedly alters the threshold for VGCC acitivation, which is almost acting like a calcium-channel blocker but which is specific for external EMF.
Here is an excerpt from an article on the CMO systems website:
I am in no way qualified to make any judgement on whether this mechanism is even possible. I have very little understanding of physics, and so cannot really have an opinion on it. I am interested to know if anyone else with a background in physics (@ark @Pierre, @Scottie or anyone else) could weigh in on this... because it seems 'too good to be true' - especially when you look at the research on biochemical parameters which I will post below
At first, I thought "just another company trying to sell some product which claims to block EMF". However, I was intrigued to learn that this device does NOT claim to block EMF. In fact, the mechanism appears to approach this problem from an entirely different angle, and so seems to be quite unique.
In short - It claims to be acting via resonance to maintain the binding of calcium ions within the proteinacous cavity, thereby modulating the VGCC threshold and preventing calcium efflux caused by external EMFs.
It has support from some of the big names in water science, including Professor Marc Henry.
For those who are not familiar with Professor Martin Pall's work + here(HIGHLY recommended), he has published several papers on the mechanisms of EMF toxicity. External EMF sources seem to activate voltage gated calcium channels inside the cell, which cause excessive calcium flooding (which has numerous downstream effects including mitochondrial damage, apoptosis, excess nitric oxide synthesis etc). Likewise, it has been proposed that these EMFs are potentially altering the biophysical properties of the calcium ion via something called cyclotron resonance (if I understand correctly):
Paper
Six studies have supported the view that calcium cyclotron resonance, has a role in producing biological effects produced by certain specific frequencies which can interact with Ca2+ ions to produce a cyclotron-like resonance (Foletti et al., 2010; Gaetani et al., 2009; De Carlo et al., 2012; Lisi et al., 2008; Pazur and Rassadina, 2009; Pazur et al., 2006). In each case, the effects involved a very specific frequency which produces the calcium cyclotron resonance and in three studies, these frequencies were shown to produce increases in [Ca2+]i levels. In the De Carlo et al. (2012) study, the calcium channel blocker nifedipine was shown to greatly lower the apparent calcium cyclotron resonance effect. This finding strongly suggests that the calcium cyclotron resonance can feed Ca2+ ions into the VGCCs, thus increasing the flow of Ca2+ions through the VGCCs into the cell following EMF exposure. The frequencies studied here for cyclotron resonance, one was close to 7 Hz and the other was close to 50 Hz, are both in the extremely low frequency range and consequently are not relevant to microwave frequency effects. The finding that only very specific calcium cyclotron resonance frequencies produce these effects is the main evidence for this mechanism.
Interestingly, many of the biological effects of EMF can be mitigated via calcium channel blockers, which strongly implicates VGCC activation being one of the primary mechanisms of cellular stress. The CMO supposedly alters the threshold for VGCC acitivation, which is almost acting like a calcium-channel blocker but which is specific for external EMF.
Here is an excerpt from an article on the CMO systems website:
The ions in our body (calcium, magnesium, potassium ions, etc.), apart from their biochemical function, also have physical properties including oscillating and rotating movements.These movements generate characteristic electromagnetic microwave signals of certain frequencies that have been accurately identified. These are an ion's signature.
The problem is that the operating frequencies of our electronic devices correspond to the spectrum of signatures of these ions, in particular Ca2+. This overlap of frequencies creates resonance or interfering effects (well known in basic physics) at the cellular level.
The Ca2+ ions in their protein cavities have characteristic rotation and oscillation frequencies within the natural magnetic field in the body which gives an overall circular movement.
The circular movement of these ions is described as having a natural cyclotron radius which is characteristic for each type of ion.
The addition of more energy to this system as happens from EMFs causes excitation of the ion and increases the cyclotron radius of the ion.
Once the cyclotron radius has been altered and increased, the ion remains at the heightened energy state and the larger radius persists.
The calcium ion (Ca2+) is held within proteins embedded in the cell membrane by biophysical forces. When the cyclotron radius is enlarged as a result of external EMFs it changes the threshold at which the Ca2+ is released from the protein binding and alters the functioning of the Voltage-Gated Calcium Channel (VGCC) through which the Ca2+ enters the cell, allowing Ca2+ to flood into the cell.
When Ca2+ enters the cells it sets off an inflammatory cascade that is the basis for a whole array of non-specific stress responses that manifest as the symptoms people commonly experience when exposed to EM pollution. We know this mechanism is correct because Prof Martin Pall discovered that these same symptoms could be prevented by giving people pharmaceuticals that block VGCCs. Chronic exposure to EMFs therefore leads to chronic inflammation which is the basis for many diseases that have been steadily rising, including cancer.
In the opposite way to how EMFs disturb the Ca2+ binding within their proteins, Compensating Magnetic Oscillators (CMO) use the phenomenon of resonance to reinforce this binding force which returns the threshold for the release of Ca2+ back to normal and corrects the regulatory function of the VGCCs. CMOs contain a crystalline solution that hold and emit the necessary resonant frequencies that were meticulously identified by the elite scientists in the Comosystems team.
I am in no way qualified to make any judgement on whether this mechanism is even possible. I have very little understanding of physics, and so cannot really have an opinion on it. I am interested to know if anyone else with a background in physics (@ark @Pierre, @Scottie or anyone else) could weigh in on this... because it seems 'too good to be true' - especially when you look at the research on biochemical parameters which I will post below