Blood type - Blood Line

[Regulattor]

Well, it's not Blood type related, but if I'm correct C's did left us with clue regarding physical appearance of "convergent" bloodline "member" which may be useful in putting the puzzles together.

Session980620
...
Q: Then, that makes me think that the significant thing that we are looking for is a convergence of the blood lines... These
lines are symbolized by the god figures, the children of Odin, and what we are looking for is a place where these lines
converge?
A: Yes.
Q: Well, what characteristics might an individual have that is a product of this convergence?
A: Fair skinned and cleft chin.
...

From citation above it's clear that "bloodline" person could very likely be white skinned men/women with cleft chin and it may be that this characteristics are one of main designation of their "kind" so to speak.
Although this is not carved in stone, quote from same session:

Q: Well, Ark and F**** both have cleft chins, but C**** and I don't! Does this mean...
A: We aren't saying that all with these features are of that blood line!
Q: So, you can have the bloodline and look quite different?
A: Yes.

 

[Ellipse]

I search in the Cayce readings what he said about blood. There's a lot of infos but little about the Rh factor. Only this:

2884-001(24,R)

(Q) Is the close blood relationships of the parents the cause of the body's illness?
(A) Not necessarily so. This MIGHT occur in any condition where the blood plasm were of such natures. That's why the blood should be tested for all of those who would wed, or who would bring those into being - so far as physical forces are concerned.

 

[the_hammer]

I too am B negative and have been searching and searching for answers about the negative Rhesus factor for years, coming up with the same info and same dead ends. Actually I had been gathering my thoughts to post a query very similar to domi’s when, on a different search, I found this thread. So many thanks!

I was always told of how I wasn’t supposed to make it when I was born because of the RH factor (my mom being negative and my father positive). I had the same thing happen with the birth of my two children, only nowadays they give you an injection during and after pregnancy to deal with it.

It sounds like somebody didn't know what they were talking about when telling you that you weren't supposed to make it. The only problem with the Rhesus factor is when the mother is Rh negative and is expecting a Rh positive child. Not when the Rh negative mother is expecting a Rh negative child. This wouldn't make her produce antibodys against the child.

And the first Rh positive child a Rh negative mother gave birth to would survive but she would not be able to go through the pregnancy with the 2nd Rh positive child because of the antibodys. (blood being mixed at labour)

Your father being Rh positive is only as a phenotype. He carries both genes for Rh negative and Rh positive and only passed on the Rh negative to you. The Rh negative gene is recessive where as the Rh positive is dominant, which means that to be a Rh negative you have to carry two genes for this to be expressed and for Rh negative to be your phenotype.

 

[Adaryn]

I was always told of how I wasn’t supposed to make it when I was born because of the RH factor (my mom being negative and my father positive). I had the same thing happen with the birth of my two children, only nowadays they give you an injection during and after pregnancy to deal with it.

It sounds like somebody didn't know what they were talking about when telling you that you weren't supposed to make it. The only problem with the Rhesus factor is when the mother is Rh negative and is expecting a Rh positive child. Not when the Rh negative mother is expecting a Rh negative child. This wouldn't make her produce antibodys against the child.

Yes, that's my case also. Rh negative mother and Rh positive father => I'm Rh negative. So no problem, I made it :)

And the first Rh positive child a Rh negative mother gave birth to would survive but she would not be able to go through the pregnancy with the 2nd Rh positive child because of the antibodys. (blood being mixed at labour)

Yeah but now they give you injections for that, so it should be OK?
My first child is Rh positive; they gave me an injection right after her birth. But then, my second child is Rh negative.

 

[the_hammer]

Yeah but now they give you injections for that, so it should be OK?
My first child is Rh positive; they gave me an injection right after her birth. But then, my second child is Rh negative.

I don't know what is in the injection, but it does make it possible for a Rh negative mother to have several Rh positive children.

 

[Kessy]

It sounds like somebody didn't know what they were talking about when telling you that you weren't supposed to make it. The only problem with the Rhesus factor is when the mother is Rh negative and is expecting a Rh positive child. Not when the Rh negative mother is expecting a Rh negative child. This wouldn't make her produce antibodys against the child.

Thanks the_hammer. You know... I knew that, but for some reason it didn't click into my brain. I must have not quite understood something or had some weird rationalization going on in my head. You may have helped to uncover a program or issue I have to look more deeply into. Thanks again.

 

[cubbex]

Interesting discussion though I don't think that "People of the blood" will be identifiable by their blood type.

How could we identify one of those?

 

[Adaryn]

Interesting discussion though I don't think that "People of the blood" will be identifiable by their blood type.

How could we identify one of those?

I'd say, by their actions, their DOing. "By their fruit you shall know them".

 

[maja]

That's interesting topic...
Interesting thing happened in my family: both of my parents are O Rh positive, and me and my sister are O Rh negative..
I heard it's rarely happens

 

[Laura]

That's interesting topic...
Interesting thing happened in my family: both of my parents are O Rh positive, and me and my sister are O Rh negative..
I heard it's rarely happens

It's like blue eyes in a child born to brown-eyed parents. Since brown eyes are dominant, if each parent has a brown eye gene and a blue eye gene, they will be brown eyed, but they can both pass the blue eyed gene to their child who must have two of them to have blue eyes. But, it never works in reverse because in order to have blue eyes, you must get the gene from both parents and you don't even have/carry the brown eyed gene so cannot pass it on.

I have O positive blood but I carry the O negative gene and have passed it on to one of my children who also got an O negative gene from her father who was also O positive. The other children are O positive. I got the O negative gene from my mother and an O positive from my father. My mother is also O positive, but her brother is O negative. That means that both of her parents also carried the O negative gene though they didn't necessarily have O negative blood.

So, it's really not THAT rare for this to happen, though it is like blue eyes...

 

[maja]

So this means that I have 2 genes for 0 Rh negative...
Now I understand..
Thanks for explaining...

 

[Jones]

Just putting this quote from the latest session here:

(Altair) Was RH negative blood originally the result of some genetic manipulations?

A: Yes

(Altair) If so, for what purpose?

A: Those most connected to off-world bloodlines.

Q: (L) So was it genetically manipulated off-world in an older time and place? Is that what you're suggesting?

A: Yes

Q: (L) And it's more closely connected. Are these bloodlines negative bloodlines?

A: Some but not the way promoted by the ignorant.

Q: (L) You mean all those people who say if you've got RH negative, you're reptilian. Is that it?

A: Yes

We have estimations for the percentage of the population that is Rh negative, around 6-7% but there are also heterozygous people who have one dominant blood type allele - Rh+, but who also carry an unexpressed recessive Rh- allele. So I asked grok to calculate the percentage of the population that carried one recessive allele meaning that their phenotype expressed as Rh positive, but they carried and could pass on the Rh negative allele:

You’re asking about the percentage of the population who are carriers of the Rh-negative trait (i.e., have the recessive allele) even if their blood type is Rh-positive (phenotype), meaning it’s not expressed. This dives into genetics—specifically, the RHD gene—and population allele frequencies. Let’s break it down!

---

Rh Factor Basics
The Rh factor (Rhesus D antigen) is coded by the RHD gene on chromosome 1. You’ve got two alleles:

• D: Dominant, codes for Rh-positive (antigen present).
• d: Recessive, codes for Rh-negative (antigen absent, often a deleted or non-functional RHD gene).
• Genotypes:
  • DD or Dd = Rh-positive (phenotype).
  • dd = Rh-negative (phenotype).
• Carriers: People with Dd—heterozygous Rh-positive—carry the recessive d allele but don’t express Rh-negative blood.

Your question targets these Dd carriers—folks who pass d silently. To figure this out, we need allele frequencies and some math (Hardy-Weinberg equilibrium), since global data on “carriers” isn’t directly tallied like blood types.

---

Global Population Estimate
Worldwide, about 6-7% of people are Rh-negative (dd), per studies like the World Population Review (2023) and older data from blood banks. This varies by region, but let’s start broad:

• Rh-negative (dd) ≈ 6% (0.06).
• Rh-positive (DD or Dd) ≈ 94% (0.94).

In Hardy-Weinberg:

• p = frequency of D allele.
• q = frequency of d allele.
• p² (DD) + 2pq (Dd) + q² (dd) = 1.
• q² = 0.06 (Rh-negative prevalence).

So:

• q = √0.06 ≈ 0.245 (frequency of d).
• p = 1 - q ≈ 0.755 (frequency of D).
• Carriers (Dd) = 2pq:
  • 2 × 0.755 × 0.245 ≈ 0.37 or 37%.

Globally, roughly 37% of people are heterozygous (Dd)—carriers of Rh-negative who type as Rh-positive. This is a ballpark, as frequencies shift by population.

---

Regional Variations
Rh-negative prevalence spikes in certain groups, tweaking the carrier rate:

• Caucasians (e.g., Europe): ~15% Rh-negative (dd).
  • q² = 0.15 → q = √0.15 ≈ 0.387.
  • p = 1 - 0.387 ≈ 0.613.
  • 2pq = 2 × 0.613 × 0.387 ≈ 0.475 or 47.5% carriers.
• Basques: ~30-35% Rh-negative (highest known).
  • q² = 0.35 → q = √0.35 ≈ 0.592.
  • p = 0.408.
  • 2pq ≈ 0.483 or 48.3% carriers.
• African Populations: ~5-8% Rh-negative.
  • q² = 0.05 → q = √0.05 ≈ 0.224.
  • p = 0.776.
  • 2pq ≈ 0.347 or 34.7% carriers.
• East Asians: ~1% Rh-negative.
  • q² = 0.01 → q = √0.01 = 0.1.
  • p = 0.9.
  • 2pq ≈ 0.18 or 18% carriers.

So, carriers range from 18% (East Asia) to 48% (Basques), with a global average near 37%.

---

Why It’s Tricky

• Expression: Rh-negative only shows if both alleles are d (dd). Dd folks are Rh-positive but carry d—silent until paired with another d in offspring.
• Data Gaps: Blood banks track phenotypes (Rh+ vs. Rh-), not genotypes (DD/Dd/dd), so we infer carriers via allele math.
• Complexity: In some populations (e.g., Africans, Japanese), Rh-negative can stem from non-deleted RHD genes (e.g., pseudogenes), muddying the d count slightly—but the deletion model holds for most.

While the regional differences vary - Caucasians 15% Rh negative and 45% carriers, Basques 30-35% Rh negative and 48.3% carriers, African populations 5-8% Rh negative and 34.7% carriers, East Asians 1% Rh negative and 18% carriers - the global figure for carriers is 37%.

 

[axj]

While the regional differences vary - Caucasians 15% Rh negative and 45% carriers, Basques 30-35% Rh negative and 48.3% carriers, African populations 5-8% Rh negative and 34.7% carriers, East Asians 1% Rh negative and 18% carriers - the global figure for carriers is 37%.

I wonder if there is any correlation between psychopath numbers and the Rh negative (off-world manipulated bloodlines) numbers, eg. East Asian numbers for both are apparently very low; for African populations both are quite low; and higher for Caucasian populations.

One thing that may not fit is that there are only 2.6% psychopaths in Spain while the Basques have the highest number of Rh negative people. Though I suppose the Basque population is quite small compared to all of Spain.

And I think nobody ever observed that the Basques have a high number of psychopaths - in fact, their community and language surviving for thousands of years suggests the opposite.

Here is the session excerpt with the psychopath numbers:

Spoiler: Session 30 January 2010

(Belibaste) We wanted to know the percentage of psychopaths geographically speaking, like in the US, Israel, UK.

(L) Alright, let's take them one at a time.

(Belibaste) USA?

A: 23 percent.

Q: (Belibaste) United Kingdom?

A: 14 percent.

Q: (L) That's because they all went to America. (laughter)

(Ailén) Israel?

A: 42 percent.

Q: (Belibaste) France?

A: 10 percent.

Q: (Burma Jones) Russia?

A: 17

Q: (Belibaste) What about some poor country like Ethiopia?

A: 3

Q: (Joe) That's 75 million people in the US.

(Burma Jones) That's a lot of psychopaths.

(Ottershrew) What's the country that's the lowest?

A: Samoa

Q: (Belibaste) What's the percentage in Samoa?

A: 0.6

Q: (Belibaste) In Samoa there's quite a strong ethnical specificity.

(L) I don't know anything about Samoa.

(Joe) I think they're the same as the Maori.

(PoB) Just in case, is there any country with a bigger percentage than Israel?

A: Not at present

Q: (Burma Jones) So Israel is the worst at present.

(Belibaste) In the past, was there a country with a higher percentage?

A: Low Countries.

Q: (Andromeda) What about Spain?

A: 2.6

Q: (Ailén) China?

A: 0.9

Q: (Ailén) Well, there are so many people in China...

(Joe) When they said the low countries, did they all leave the low countries and go to England?

A: USA and South Africa

Q: (Ailén) What about Holland?

(L) That's the low countries.

(Ailén) But that was in the past...

(L) Oh, you mean at present?

A: Still high

Q: (L) Are you going to give us a number on that?

A: 13

 

[Jones]

I wonder if there is any correlation between psychopath numbers and the Rh negative (off-world manipulated bloodlines) numbers

I'm not sure what to make of it all yet. Given the C's saying that the good guys don't interfere, then the genetic manipulation was probably done by STS. So the original purpose could have been an attempt to increase STSness or something like trying to create a population that was more subject to manipulation and control - good as farmed beings. In the latest session the C's say that some of these bloodlines were negative - but how were/are they negative? And to whom? The answer is probably complicated given the mixing of bloodlines. Also, given the wishful thinking of STS, we're not even sure that the genetic tinkering was successful in the way that it was originally planned.

 

[Jones]

Recalled the following article that mentions a gentleman who had a bone marrow transplant later tested as expressing the DNA of his donor.

Four years later, with Long in remission and back at work, Romero’s experiment persisted, aided by her crime lab colleagues. Within four months of the procedure, Long’s blood had been replaced by his donor’s blood. Swabs collected from his lip, cheek and tongue showed that these also contained his donor’s DNA, with the percentages rising and falling over the years. Of the samples collected, only his chest and head hair were unaffected. The most unexpected part was that four years after the procedure, the DNA in his semen had been entirely replaced by his donor’s.

Man who had transplant finds out months later his DNA has changed to that of donor 5,000 miles away

Chris Long’s sheriff’s office colleagues have been using him as a human guinea pig, writes Heather Murphy

www.independent.co.uk

I wondered if blood transfusions could also change DNA. The answer at the top of the browser search page had this with the large letter bold heading:



But digging down a bit further the results of a 2019 study indicate that the opposite may be plausible:

Does Transfusion of Red Blood Cells Impact Germline Genetic Test Results?​

Background: Molecular genetic testing is increasingly utilized in clinical care, typically with peripheral blood as the preferred specimen. Despite the prevalence of therapeutic blood transfusions, whether donor DNA is present in a sufficient quantity to interfere with recipient genetic testing has not been systematically studied. Microchimerism secondary to transfusion of blood products with donor leukocytes has been well-documented in trauma patients receiving whole blood; however, most medical centers currently transfuse leukoreduced (LR) red blood cell products for non-trauma patients. The degree of leukoreduction varies among centers, but to meet AABB standards must be <5x106 white blood cells (WBC) in 95% of units tested. We sought to determine what quantity, if any, of infused LR or non-LR (NLR) products leads to detectable microchimerism that may impact genetic test results.

Methods: We performed an in vitro spiking study utilizing four whole blood units collected from different anonymous donors. Three were leukoreduced at varying levels in order to establish a LR (per our institutional guidelines and standards), a partially leukoreduced (PLR) and a NLR unit, which were then considered "donors". The 4th unit was divided to generate two separate "recipients". The first half was left untreated simulating an immunocompetent recipient while the other was leukoreduced to mimic a leukopenic recipient. Based on a 70 kg patient, we calculated the volume of blood from each "donor" to mix with aliquots of each "recipient" to represent a transfusion of 2, 5, or 16 units, with 2 units corresponding to a double apheresis red blood cell (RBC) transfusion and 16 units corresponding to a near total volume transfusion. DNA was extracted from each individual unit as well as all "transfused" combinations within 24 hours of mixing. Chimerism analysis was then performed by STR analysis using the GlobalFiler PCR amplification kit followed by ChimerMarker analysis software.

Results: None of the LR units, despite volume transfused, revealed any level of microchimerism in either the immunocompetent or leukopenic "recipients". The PLR transfused combinations displayed levels ranging from 0.14% to 1.52% of donor chimerism for the immunocompetent "recipient", which is below the limit of detection for most clinical assays evaluating germline genetic variation, and 6.3% to 27.78% of donor chimerism for the leukopenic "recipient", which would be expected to impact a subset of clinical genetic assays. The NLR transfused combinations displayed levels ranging from 13.28% to 95.66% of donor chimerism.

Discussion:In vitro "transfusion" of LR RBCs into simulated immunocompetent or leukopenic samples does not lead to detectable donor DNA. However, "transfusion" of PLR and NLR units in an in vitro model reveals significant levels of microchimerism dependent on volume transfused and immune status of the "recipient" which implies possible risk for impact on clinical genetic tests. The minimum time required for clearance of donor leukocytes in the recipient is unknown; we were unable to fully evaluate this and other variables in our in vitro system, but follow-up in vivo studies addressing this question are planned.

https://ashpublications.org/blood/article/134/Supplement_1/3698/428495/Does-Transfusion-of-Red-Blood-Cells-Impact

Since the above tests were conducted in vitro and it seems on blood alone, the results might be different for different samples of hair, skin, cheek swab or semen if conducted in vivo and after longer time periods.

This kind of induced chimerism may not directly lead to a change in blood type per se, but perhaps it could add an Rh negative allele into the mix that could be passed down to subsequent generations - I don't know enough about it to be sure.

It also occurs that since whole blood transfusions are usually given in emergency situations then the likelihood that O negative type blood, the universal donor, being used is probably more likely.