Great book on neurotoxic aluminum

I think his current recommendation is to consume at least a half liter of "silica" water per day. Fiji water is a good choice - I use a half liter to make morning coffee. He warns against other forms of silica as ineffective.

Some say that beer can be effective:

The chemical affinity of silicic acid for aluminium (AI) has been shown to reduce the bioavailability of AI in studies of human gastrointestinal (GI) absorption. Investigations were carried out to ascertain whether or not similar interactions may also enhance the renal excretion of AI by assessing the urinary output of both elements. Healthy individuals given monosilicic acid as naturally found in beer, excreted the majority of the silicic acid content (mean 56 percent) within 8 hours, concomitant with a significant increase in AI excretion (P < 0.05). Ingestion of increasing doses of silicic acid resulted in dose related increases in excretion of Si. Excretion of AI reached a maximum and then declined, consistent with depletion of AI body stores. This was confirmed using the 26AI isotope. The low serum but high urine concentration of Si suggests that if AI and Si interact to form an excretable species they do so in the kidney lumen such that Si limits the reabsorption of AI. Silicic acid's effect on the depletion of aluminium stores and reduced GI absorption suggest its addition to municipal water supplies may be a low risk public health measure to reduce the AI burden in the general population.



Thus, we found no evidence for the mobilisation of Al from body stores following ingestion of orthosilicic acid. In contrast, Bellia et al. [20] observed a significant concomitant increase in urinary elimination of Al (or an unlikely absorption of 26% of the ingested Al (72.1 mg)) following the intake of beer which contained silicic acid. They suggested an interaction, in the proximal convoluted tubule of the kidney, between Si and Al leading to their co-elimination. It is not clear why our results differ, but a number of explanations should be considered. First, a small peak of Al may have occurred in urine following ingestion of ortho-silicic acid that would only be apparent in urine collections of much shorter time intervals (e.g. in catheterised subjects). However, Bellia et al. [20] also used 8 h collection periods for urine and were able to observe a difference. Secondly, alcohol may increase gut permeability as it is known to affect lipid bilayer permeability [42], which would then increase Al uptake from beer. However, a 26% increase seems unlikely. Thirdly, Si and alcohol may have a synergistic effect on the excretion of Al, which is not observed with alcohol [20] or Si alone (Fig. 2). Fourthly, a component of beer other than alcohol or silicic acid may be responsible for the excretion of Al from body stores. Finally, the body pool of Al of the subjects in the Bellia study [20] may have been higher, as volunteers were older than the subjects in this study.



 
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