How it really happened

Hope

Padawan Learner
Hi everybody,

I am an ex forum member. My past names were Gee, Infiniteness, and some other names that I can’t remember no longer. I deleted my account in mid-2012 because of frustration and probably due self-importance as well, after causing even more damage to my health. I am writing this for feedback and maybe this can help others not make some of the mistakes that I made as well. I am just going to tell you what has happened, trying to be truthful and accurate as possible.

Growing up I had some delays in development it seems like. But things really started going downhill for me when I was in 8th grade. I started devolving problems with OCD, paranoia, hypochondria, and anxiety. As time went by things kept getting worse for me mentally. In 12th grade I started having symptoms of paresthesia, which drove my hypochondria into overdrive. Which I now know was caused a great deal by gluten/grains, dairy, not being breastfeed, all that metal in my mouth, etc...

At the time I was getting into “conspiracy theories” and happened to stumble upon MMS. Being gullible and naive I started taking it in February 2009. I think I took it for a couple weeks, I do remember going up to 15 drops. But I don’t really remember how much and for how long I took it. One of the worst mistakes I’ve made, as it probably did a lot of damage to my gut and making my gluten allergy so much worse. I really started to deteriorate after that.

Then in September 2010 as I was trying to go gluten and dairy free, I made the mistake of substituting wheat with corn. The corn was GMO, which caused such a severe reaction (BT toxin), that it lasted more than 9 months. Had a laundry list of symptoms, had strong suicidal ideation as well (Sorry for making all that noise on the forum during that time period). But I didn’t think much of it because my mom would make roti from it since as long as I can remember.

So in late 2011 I was in the process of trying to eat paleo/keto and ended up going on a really bad binge of eating junk food. After a couple of weeks of not being able to stop myself, I had a major flare up and my vision deteriorated even further, went from bad to worse. I would tell myself every day that I needed to stop eating gluten while on the binge but just kept eating and eating. I never thought it cause such a reaction!

After a couple of months of freaking out, I decided to go on the Paleo diet. I didn’t come back here because I felt like I already bothered you guys enough plus I felt like a failure. I eliminated all the grains, dairy, soy, GMOS. But after couple months of being on it. I started to developing some new and serious symptoms. I started urinating frequently, urine became cloudy, more bubbles, change in voice tone, loss of buttocks hair, and worst of all I developed sexual dysfunction! Now that I look back at it, I believe I was eating too much protein, mostly grass fed ground beef and eggs. At the time I thought I had damaged my kidneys especially after poisoning my body with MMS and GMO Corn but after going to many doctors and having many tests done (all tests came back normal), I’m not sure what I did?! Anybody have an idea what might of happened? I deleted my account in the summer of that year because I was sure it was kidney damage and felt like a total failure.
So for the next two years I just went on a gluten free and gmo free diet. After the first year of developing these symptoms I just gave up, after the doctors could find nothing wrong. I then started smoking cannabis heavily. From summer of 2013 to the end of summer 2014. I usually smoked every day, multiple times a day. I quit smoking on Sep 6th 2014 and have been sober since. And yes withdrawal symptoms are real! Felt like I was losing my mind for the first two weeks, but I feel normal now.

Recently I got a new job that pays well. Only downside is that I have to work graveyard shifts but am hoping to switch to days sooner or later. My symptoms have improved gradually over the past couple of years but I continue have neurological flare ups. Which I know are due to the rice and some other foods that I still eat like potatoes. I have recently eliminated all processed foods from my diet, starting using Himalayan salt and am in process of only using gluten free soaps and shampoos. I need to get Keto adapted, its probably my best bet at stopping this neurological damage and maybe reverse some of the damage I have done. I think about it going Keto all the time but I am afraid I might screw up again. I've seen three naturopathic doctors over the years but it was just a waste of time and money I feel like, I felt like they didn't have the necessary knowledge to help me get Keto adapted.

So it up to me to do what needs to be done. I’ve read Life without bread, Keto adapted, am currently reading Primal body primal mind and rereading Dr. Segura Sott summery book. I have replayed the events in my head over and over and am baffled at how I did all of this. If I had just an ounce of common sense, none of this would have happened. I only have myself to blame, but I’m done being stuck in the past, pitying and loathing myself. I feel like the post is incomplete or maybe too long but I am just going post it because I have been putting it off for months now.

Thank you for reading.
 
Welcome back AD, it looks as if you have hit rock bottom.
Now the only way to go is upwards and onwards!
Blessings. And good luck with the diet.
 
Hi AD,

I am glad you decided to post. It's good to see you back. It sounds like you are on the right track with your diet changes. The keto diet is incredibly healing. Welcome back! Your welcome message is below. :)

Don't forget to check out Tips and advice for Newbies and the Forum Guidelines.

You might also want to read through the Diet and Health and EE-Breathing Program threads as well as the Recommended books: List and Guide. You also might be interested in the Knowledge and Being video made by Laura. It serves as an introduction to some of the concepts discussed here on the forum.

In the meantime, it's a good idea to bookmark the Cassiopaea Esoteric Glossary as it is a very useful reference that can be used when you encounter concepts/terminology that you may not be familiar with. It should be consulted before posting questions on the forum.

Note that there are some "off limits" areas to the forum that are restricted to all but active forum participants, and we ask you to please keep the noise down. You will also not be able to edit or delete your posts until you reach a minimum of 50 posts. In that respect, it pays to use the "Preview" button prior to posting your message so that you can see how your post will look to others.

Finally, there is so much information on this forum that before beginning a new thread or asking a question on a subject, it is recommended to use either the quick search function at the top of the page or the more advanced search function on the menu function.

Please note that on each section of the forum, you will see a sticky topic at the beginning of the list of topics. This sticky topic called "Important threads - Name of the section of the forum" contains all the important threads that should be read by all members.

For members interested in delving deeper into the material, check our Comprehensive Guide for the Serious Reader.

Looking forward to your participation.

P.S.: Also, if you're not aware of it, the Signs of the Times news page is an excellent place to get your news. ;D
 
First of all i wish you a good luck with work on yourself. Be strong and try to fight with yourself when bad thoughts came in your mind.
I dont know if you are practicing EE but it makes a wonderful thing for me.

Just pure reading nor means that you have a right knowledge. Real knowledge is when you are applying that informations. First on yourself. You must understand how your machine is working like G said.Then you can try to fix it. We are all trying to understand our own machines and trying to repair whats can be repaired and to be aware and consciousness of every action.Sometimes its very hard , sometimes its easier but you must keep doing it.
10 years ago i weight 110 Kg and i eat a tons of bread, and cakes and all kinds of sweets and alcohol. Even then i know that its bad but i was still doing.Until one day my health was in serious problems. Then i really understand that it is a bad lifestyle. And after that shock i started work to improve my health and i managed to get my weight from 108 to 72Kg in just one year. I quit eating bread, sugar, all things that contain gluten.
And still it was not a perfect situation. I still have to examine my machine and try to fine tune it with a lot of supplementation, vitamins, physical activity, rest and sleep regulation. The really hard for me was the stress management , but with EE i started to improve in that area too.

What i want to say with all this above is that only thing that you need is a strong will right now. You have all needed information you just have to apply it and convert it to knowledge.

If that knowledge is real one you have to apply it and be real about results from it. If results are not ok, then you have to change something , if they are ok you are on the right track.
Network and listen how other people are trying to understand their body. We all make a lot of mistakes during our lives. Dont blame youself. Just learn from that mistakes and keep going and follow the work. It the best way to improve yourself.

I wish you all the best on your way
:rockon:
 
Welcome back AD, it sounds like you've gone through some pretty heavy learning.

You might want to check out the Diet podcast that just started up: http://www.blogtalkradio.com/sottradionetwork/2015/01/12/the-health-and-wellness-show--12-january-2015
Also (as most people aren't eating enough fat so are eating too much protein and/or craving carbs) have a look at the fat bomb recipe thread. I now have a huge chunk of my diet in that form :D

Some SoTT articles that may help too (although go super slow and steady if you give it a go!)
http://www.sott.net/article/229352-Tips-Tricks-for-Starting-or-Restarting-Low-Carb-Pt-I
http://www.sott.net/article/230561-Tips-tricks-for-starting-or-restarting-low-carb-Pt-II

Some possible points of data from what you've described of your health conditions.
http://ndt.oxfordjournals.org/content/26/7/2137.full
[..]
Conclusions.

CKD results in marked down-regulation in the expression of folate and thiamin transporters in the intestine, heart, liver and brain. These events can lead to reduced intestinal absorption and impaired cellular homeostasis of these essential micronutrients despite their normal plasma levels.

Chronic kidney disease (CKD) has emerged as a major public health problem worldwide [1]. CKD is associated with an increased risk of cardiovascular disease, neurologic disorders, malnutrition and progression to end-stage renal disease. Vascular and neurologic complications in particular remain an important source of morbidity and mortality in this population [2,3].

The water-soluble vitamins are a group of structurally and functionally unrelated compounds that share the common feature of being essential for normal cellular function, growth and development. Folate and thiamin (vitamin B1) are two members of this family of micronutrients. Folate is required for the synthesis of pyrimidine and purine nucleotides (precursors of DNA and RNA, respectively) and for the metabolism of several amino acids including homocysteine [4,5]. Folate deficiency leads to disruption of folate-dependent metabolic pathways, effects that lead to the development of clinical abnormalities ranging from anemia to growth retardation [3–5]. Thiamin serves as a cofactor for multiple enzymes involved in critical metabolic reactions which relate to energy metabolism [6]. Because it bridges the glycolytic and the pentose phosphate metabolic pathway, thiamin is also critical for creating chemical reducing power in cells [7]. Therefore, thiamin is thought to play an important role in reducing cellular oxidative stress [7–11]. Thus, low intracellular levels of thiamin lead to impairment in energy metabolism and a propensity for oxidative stress, which are a common finding in CKD. At the clinical level, thiamin deficiency leads to a variety of abnormalities that include neurologic (neuropathy, Wernicke-Korsakoff syndrome) and cardiovascular (e.g. peripheral vasodilatation, biventricular myocardial failure, edema and potentially acute fulminant cardiovascular collapse) disorders [7–11].

Human and other mammals cannot synthesize folate and thiamin, and thus, they depend on the intestinal absorption of these micronutrients from exogenous sources. Once absorbed, folate and thiamin are distributed throughout the body via the circulation. Intestinal absorption of folate and thiamin and their uptake into different cell types have been the subject of intense investigations over the past three decades. It is well known now that the reduced folate carrier (RFC) and the proton-coupled folate transporter (PCFT) are major folate transporters in intestinal and other mammalian cell types [12–14]. For thiamin, the thiamin transporters 1 and 2 (THTR-1 and THTR-2) were shown to be the main transporters in intestinal epithelial cells and in other cell types [15–18]. Furthermore, studies have shown that the expression of these vitamin transport systems in different tissues is highly regulated and influenced by extracellular and intracellular conditions [15–18].

In view of the association of CKD with oxidative stress, neurological disorders, myocardial dysfunction and hyperhomocysteinemia which can also be caused by folate and thiamin deficiencies, information on the effect of CKD on expression of their transporters in relevant tissues would be of interest. The available data on possible effects of uremia on expression of folate and thiamin transporters are limited. The present study was designed to explore the effect of CKD on the expression of folate and thiamin transporters in key tissues using an experimental animal model of CKD. We focused on these tissues since they are important for homeostasis (intestine), storage (liver) and utilization of these vitamins.
[..]

Discussion and Conclusion

Some of the clinical features of CKD such as hyperhomocysteinemia, uremic neuropathy, easy fatigability and oxidative stress resemble those found in the vitamin deficiency states. While plasma folate and thiamin levels are reportedly normal in CKD patients, the latter observation raises the possibility that CKD may be associated with localized (functional) deficiency of these and other vitamins [19–23]. In this context, earlier studies by our group have demonstrated significant reductions in the intestinal absorption/uptake of several water-soluble vitamins including folate, biotin, riboflavin and pyridoxine in the rat model of CKD [24–27]. In addition to affecting intestinal transport of these nutrients, uremia is known to impair uptake and transport of certain drugs and down-regulate organic anion transporters in the liver, intestine and brain in rat model of CKD [28–30]. In light of these findings, we sought to assess the effect of CKD on folate and thiamin transporters in selected organs. The study revealed a significant down-regulation of the folate transporters RFC and PCFT in the small intestine of CKD animals compared with sham-operated controls. Down-regulation of intestinal folate transporters found in the present study provides the mechanism for the reduction of intestinal uptake of folate in the uremic rats shown in our previous studies [24]. Down-regulation of intestinal folate and thiamin transporters shown here and reduced intestinal folate absorption shown previously were paradoxically associated with no significant change in the plasma thiamin or folate levels in the CKD animals compared with the sham-operated control rats. The observed normality of plasma folate and thiamin levels in the CKD animals is consistent with the results of the published studies in humans [19,20]. This phenomenon cannot be attributed to dietary factors since the amount and composition of food consumed by the CKD and control animals were similar. Likewise, a possible reduction in the volume of distribution of these products is an unlikely candidate since the opposite would be expected due to volume expansion in humans and animals with advanced renal insufficiency. However, diminished uptake and utilization of these vitamins by peripheral organs and tissues can potentially account for their normal plasma concentrations despite reduced intestinal absorption. This supposition is supported by the observed down-regulation of folate and thiamin transporters in the liver, brain, left ventricle and hepatic MFT in the CKD animals.

Folate plays a critical role in the purine and pyrimidine biosynthesis (precursors of nucleic acids), in the metabolism of several amino acids (including homocysteine) and in the initiation of protein synthesis in mitochondria [4–6]. Folate deficiency leads to both chromosomal DNA injury and mitochondrial DNA mutations [4–6,31], which have been collectively associated with mitochondrial dysfunction, membrane depolarization, increased reactive oxygen species (ROS) production and premature cell death [31,32]. It is well established that CKD is associated with up-regulation of ROS-generating machinery, increased generation of ROS, oxidative stress and inflammation [23]. Therefore, a potential decrease in cellular uptake of folate in such tissues as liver, heart and brain and a potential decrease in folate uptake by the mitochondria may contribute to the prevailing oxidative stress in CKD. It has been shown that mitochondrial folate deprivation can induce oxidative stress by promoting cytochrome C oxygenase dysfunction, membrane depolarization and super-oxide overproduction. In this context, folate serves as an antioxidant at the mitochondria level and helps to ameliorate the mitochondrial oxidative decay elicited by pro-oxidants [31,32]. Given the important role of liver in storage and metabolism of folate, a potential defect in mitochondrial folate transport in this organ may lead to mitochondrial dysfunction, heightened ROS production and oxidative stress in CKD [31,33,34].

The findings of the present study may help explain the failure of clinical trials of folate supplementation to improve mortality and cardiovascular outcomes in hyperhomocysteinemic patients with CKD {It's unlikely the folate was in a reduced form - standard folic acid would most likely agrivate the condition. Best to avoid vegetables and suppliments with Folate in and eat some liver instead}. The rationale leading to the expectation that folate should have been beneficial arises from the fact that folate is also involved in the methionine metabolism pathway by serving as a substrate for the methionine synthase [35]. Reduced folate availability can result in hyperhomocysteinemia that has been associated with increased risk of atherosclerotic cardiovascular disease as well as neurological complications [36–39]. Patients with CKD frequently exhibit significant hyperhomocysteinemia [39,40]. While some have suggested that strategies aimed at reducing homocysteine level in CKD may reduce the burden of atherosclerosis and improve mortality, a randomized clinical trial of folate supplementation failed to change overall mortality and cardiovascular outcomes in this population [41]. Furthermore, folate supplementation failed to normalize homocysteine level in most patients with CKD in this trial [41]. The down-regulation of folate transporters and their decreased protein abundance in the liver shown here may, in part, account for the failure of folate supplementation in normalizing plasma homocysteine levels in CKD population.

Thiamin plays a fundamental role in cellular metabolism and energy production, principally via thiamin pyrophosphate, which serves as a coenzyme required for normal cellular function, growth and development [9–11]. Furthermore, thiamin plays a role in blunting ROS generation via its capacity to bridge the glycolytic and the pentose phosphate metabolic pathways [9–11]. Insufficient amounts of dietary thiamin can lead to a variety of clinical consequences that include neurologic and cardiovascular dysfunctions [42–44]. In this study, we found significant down-regulations of the thiamin transporters THTR-1 and THTR-2 in all tested tissues and decreased protein abundance in the liver of CKD animals when compared with sham-operated controls. We speculate that down-regulation of thiamin transporters will result in diminished intracellular thiamin levels and will contribute to the CKD-associated oxidative stress.

CKD results in reduced exercise capacity which has been attributed, at least, in part, to the associated anemia of CKD. The advent and widespread availability of erythropoiesis-stimulating agents (ESAs) has revolutionized the treatment of CKD-induced anemia. Contrary to expectation, correction of anemia with ESAs has had minimal impact in restoring CKD patients’ exercise capacity [45]. Because thiamin is an important factor for glucose metabolism and energy production, possible limitation in cellular thiamin availability can adversely affect energy metabolism [43,46]. In addition, thiamin deficiency has been reported to cause cognitive impairment and findings similar to Alzheimer’s disease [47]. It is of note that thiamin serves as a cofactor for transketolase (a key enzyme in the non-oxidative branch of pentose phosphate shunt pathway) whose deficiency is known to cause peripheral neuropathy [42,46,48]. Peripheral neuropathy is a common complication of uremia, and uremic peripheral neuropathy has been linked to dysregulation of transketolase [49]. Given the critical role of thiamin in regulation of transketolase activity, we speculate that the observed down-regulation of thiamin transporters in the neuronal tissue shown in the animal model of kidney disease contributes to the pathogenesis of uremic neuropathy.

It is also interesting to note that CKD results in a significant reduction in the expression of mitochondrial thiamin transporter (SLC25A19). In light of the fact that thiamin is an essential cofactor for three enzymatic complexes within the mitochondria (the pyruvate dehydrogenase complex, the branched chain keto acid dehydrogenase complex and the alpha-ketoglutarate dehydrogenase complex) and the fact that mutations in this transporter are believed to cause progressive polyneuropathy and bilateral striatal necrosis, our findings may have significant clinical relevance in regard to the peripheral neuropathy observed in CKD patients [50].

In conclusion, the present study demonstrated the association of CKD with down-regulation of folate and thiamin transporters, which can potentially contribute to various complications of uremia by limiting bio-availability of these essential micronutrients at the tissue, cellular and organelle level.

So checking the folate connection
http://www.ncbi.nlm.nih.gov/pubmed/23595572
MTHFR, MTR and MTRR polymorphisms and risk of chronic kidney disease in Japanese: cross-sectional data from the J-MICC Study.

Abstract
PURPOSE:
Chronic kidney disease (CKD) is well known as a strong risk factor for both of end-stage renal disease and cardiovascular disease. To clarify the associations of MTHFR, MTR, and MTRR polymorphisms with the risk of CKD in Japanese, we examined this association among Japanese subjects using cross-sectional data.
[..]
CONCLUSIONS:
The present study found a significant association between the subjects with the T/T genotype of MTHFR C677T polymorphism and the elevated risk of CKD, which may suggest the possibility of the risk evaluation and prevention of this potentially life-threatening disease based on genetic traits in the near future.

Kidney problems are going to cause sodium, potassium and other trace minerals to be depleted.

Also as you mentioned amalgam fillings - MTHFR mutations tend to result in extremely sluggish detox systems, and mercury tends to deplete thimine/minerals.
If you do decided to do any detox protocols go Very cautiously as they will stress the kidneys.
*edit to add* also be very careful with NAC, not only may it push things too hard, if you have the mutation it may not be good for you at all.

I'm working on testing/refining some protocols for MTHFR polymorphisms, which include things for protecting/healing the kidneys. Although it could simply be just mercury/stress, developmental delays etc hint at this type of polymorphism or chronic folate deficiency.
 
Welcome back AD. There are some great replies here for you. Just to add that when i went on a paleo diet following the advice of other forum members that after a few months many of my biggest problems like anxiety and pain etc pretty much dissapeared. I am just in the process of getting my diet back under control after a slip. EE has also been a tremendous help for me.
 
I'm glad you came back AD. I went through a bad spell and quit sott and the forum twice. There are some very patient people here. I have trouble writing on here and than when i do i wonder if i am posting or making noise. I think if we just do our best and keep working and networking we will learn to become comfortabe and learn how to use the forum effectively. There are a lot of great links to information above i think many of us will find usefull. Welcome back AD. Good luck on the paleo. Tarri
 
Welcome back.

Everything is a part of learning,whether its about ourselves, nature of the world or psychology.

We need to always be vigilant regarding our thoughts,actions etc.

We as humans, are all in this together, so hopefully we can use our willpower towards ourselves and others, to head towards STO= service to others

Everyone is all doing well,keep up the good job.

I am happy and enjoy each n every day,because it gives me time to think and learn.

Hopefully everyone has a nice weekend :)
 
Thank you guys for the replies, I will be responding as soon as I have time. I work 12 hour shifts, 3 to 4 nights out of the week. So those days all I have time to do is work and sleep. :rolleyes:
 
Thanks guys for the feedback, really means a lot to me. I am going to switch over to eating wild rice in the next couple of days and ditch the white rice, also nightshades. Eat two to three servings of wild rice a day with vegetables and meats. Will still will be eating high carb but I think its best until I feel comfortable enough to start the transition into getting Keto adapted. I also try not eat too much protein because I still feel like I might have done damage to my liver/kidneys. So I will be staying away from grains, diary, GMOS, processed foods and I cook all my food in organic butter.

Hi Redfox,

Thanks for the article, I wont be doing any detoxing until I get fully Keto adapted. I don't any take supplements at the moment. I am seeing a mercury free dentist but for now I'm just getting new tooth decay and missing/broken fillings taken care of. Waiting until I get Keto adapted and then start the process of removing all of the amalgams.

I just gotta make sure I do this right, so making sure I get the reading done and ease myself into it.

Thanks once again.
 
Hi everyone,

I just wanted to give an update on how things are going for me. I have eliminated the white rice, potatoes, and tomatoes about two weeks ago.I have replaced it with wild rice and other healthier veggies. I really don't think I can tolerate the wild rice too well, sometimes quite a bit of it comes out undigested in my stool. So I have ordered digestive enzymes from NOW foods and will see how much that helps out. Along this same time I have been feeling pretty edgy and anxious at times, I think maybe I might be having withdrawal symptoms from eliminating the white rice etc??

I have lost quite a bit weight since getting my new job and making these new dietary changes, down to 118lbs/height 5'10. Feel like an anorexic, but I have always been a skinny! I am going add some more foods into my diet like beans and eat more, I have also starting weight lifting again so that should help me gain some weight, until I go Keto. I need to start practicing how to make the fat bombs and bone broth, make sure I know how to make it before I start the transition. Just been lazy and putting it off. :rolleyes:

I also noticed a couple of days ago that my right thumb has a bit of a orangish color to it, like a stain. I think its due to all the sweet potatoes I eat or from the cigs. I doubt its anything serious, but if doesn't go away in a couple of days time. I will go see the doc.

Just trying to take it one step at a time, thanks for reading.
 
As Constantine said, we need to pay attention to your machine (your body)
It is interesting that once a year we have to perform checks on his car, before that it regularly serviced, swapping out everything there is to modify, change the oil, if you should buy a new part we make sure that everything is OK.
When it comes to our body, it informs us when sick, exhausted ... but most of us does not change, some of your eating habits, if you need regular check-ups ..., it only makes your body to the limit.


I am glad that you are on time taking care of your health, be insistent, some parts can not change as the car. :)
 
Hi AD, and welcome back!

in my experience, the first thing to do is to take care of the digestive system, that is intestines and stomach. Nora Gedgaudas and others recommended probiotics (first thing in the morning, on an empty stomach with some greasy food), after 0.5 hour you can have breakfast. between meals you can try to have 1 teaspoon of L glutamine dissolved in warm water which can regenerate the gastrointestinal mucosa and depleted secretory IgA levels (immune defense) of the intestines. L glutamine is not recommended when somebody has cancer.
If you're feeling OK, you can increase up to 3-4 grams per day, taken on empty stomach.

You said you have digestive enzymes, take it right after the main meal. Attention, I just heard there was a problem here - one manufacturer had the idea to put chilly pepper in one of this "super enzyme" brand which caused severe nasal bleeding to one of my acquaintance, so check first !!!

All serotonin is produced in the gut, so mood issues are related to gastrointestinal health. If you don't have it restored, there is useless to take the best supplements, as they will not be absorbed properly.

I hope this will be of help,
best wishes
Joy
 
AD said:
Hi everyone,

I just wanted to give an update on how things are going for me. I have eliminated the white rice, potatoes, and tomatoes about two weeks ago.I have replaced it with wild rice and other healthier veggies. I really don't think I can tolerate the wild rice too well, sometimes quite a bit of it comes out undigested in my stool. So I have ordered digestive enzymes from NOW foods and will see how much that helps out. Along this same time I have been feeling pretty edgy and anxious at times, I think maybe I might be having withdrawal symptoms from eliminating the white rice etc??

I have lost quite a bit weight since getting my new job and making these new dietary changes, down to 118lbs/height 5'10. Feel like an anorexic, but I have always been a skinny! I am going add some more foods into my diet like beans and eat more, I have also starting weight lifting again so that should help me gain some weight, until I go Keto. I need to start practicing how to make the fat bombs and bone broth, make sure I know how to make it before I start the transition. Just been lazy and putting it off. :rolleyes:

I also noticed a couple of days ago that my right thumb has a bit of a orangish color to it, like a stain. I think its due to all the sweet potatoes I eat or from the cigs. I doubt its anything serious, but if doesn't go away in a couple of days time. I will go see the doc.

Just trying to take it one step at a time, thanks for reading.

Introducing beans to your diet will not make your digestion better.It may be the opposite. That is my experience. Maybe you can make fat bombs and the most important is the bone broth. I think its the best thing that you can offer to your body.

You have mentioned that you have maybe damaged liver/kidney. It better to check that.If there is some damage the consult a doctor before taking L-glutamine.
Increasing fats in your diet can help you in your weight lifting too, since your body will be forced to burn fats for energy more rapidly. And be careful with too many protein consumption. If you are increasing protein consumption then increase the fat consumption too.
 

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