Arctodus
Jedi
This is a response to commenter Ruth regarding this article.
http://www.sott.net/articles/show/197404-The-Flu-Epidemic-All-Fatal-Ukraine-Cases-at-GISAID-Have-RBD-D225G-Mutation
From Commenter Ruth.
"Recently saw a TV program about a geneticist who was studying human gene mutaion Delta 32. He discovered this interesting anomaly whilst studing a haemophiliac who had been given multiple injections of factor 8 for blood clotting (which at the time would have contained HIV). This haemophiliac never developed HIV because the gene mutation seemed to 'bounce' the virus off the cell receptor.
He linked its increased frequency in the population to the Plague which killed so many people in Europe 700 years ago. It's kind of odd that scientists are now querying whether Bubonic Plague was actually a virus.
[Link]
Its also odd that the highest frequency of this genetic mutation which is resistant to (some?)viruses should occur in the same areas that this Flu mutation now is (RBD D225G): Scandinavia and Russia. It all just seems a bit strange."
I would suggest reading this article "FACTOR VIII, HIV AND AIDS IN HAEMOPHILIACS: AN ANALYSIS OF THEIR RELATIONSHIP"
http://www.virusmyth.com/aids/hiv/ephemophilia.htm
posted section of the Abstract.
"Introduction
Currently, it is accepted that many patients with haemophilia have become HIV infected and/or developed the AID clinical syndrome as a direct result of the transfusion of factor VIII preparations contaminated with this particular virus. That this is indeed the case requires proof:
1. of the existence of a unique, infectious retrovirus, HIV. (For a critical discussion of this issue see Papadopulos-Eleopulos, 1988, Papadopulos- Eleopulos et al., 1992, Papadopulos-Eleopulos et al., 1993a, Papadopulos- Eleopulos et al., 1993b);
2. of the existence of HIV in factor VIII preparations;
3. of the existence of HIV in haemophiliacs;
4. that HIV is necessary and sufficient for the decrease in T4 cells observed in haemophiliacs;
5. that HIV and a decrease in T4 lymphocytes are necessary and sufficient for the development of the clinical AID syndrome.
Factor VIII and HIV
Since factor VIII is made from plasma, as a first step in proving contamination of this blood product with HIV, evidence must be presented that infectious viral particles with morphological characteristics attributed to HIV, are present in the plasma of "HIV infected" individuals. Then it must be shown that HIV can survive (a) the time between blood collection and freezing of plasma; (b) the freezing and thawing itself; © the process of manufacturing factor VIII from thawed plasma. In other words, as Jay Levy succinctly expressed in 1989, it is "important to know whether retroviruses could survive the preparation involved in producing Factor VIII concentrates. Otherwise, AIDS in many haemophiliacs [a minority may have other risk factors] could not be explained" (Levy, 1989).
HIV in plasma
To date, there is no evidence of the existence in human plasma of particles with the morphological characteristics attributed to HIV even though the plasma of at least some "HIV infected" individuals is claimed to contain such particles. Thus Levy, whose team reported most often on the relationship between HIV and factor VIII wrote in 1988: "Human Immunodeficiency virus in plasma or serum has been found in about 30% of specimens from seropositive persons, generally at a concentration of less than 10 IP/mL12" [IP=3Dinfectious particles] (Levy, 1988). Reference 12 cited by Levy is a paper which he published in collaboration with Barbara Michaelis but this paper does not contain a description of the method used to show that (a) HIV seropositive (non-haemophiliac) plasma was infected with "HIV particles"; (b) HIV was "present in low titers"; (c ) the particles were "infectious". Commenting on his and his colleagues' findings Levy wrote: "These studies demonstrate further that not all seropositive individuals have virus recoverable from their PMCs and that isolation from serum is not a common event" [PMCs=peripheral blood mononuclear cells] (Michaelis & Levy, 1987). "Thus, cell-free virus in body fluids is unlikely to be a meaningful source of HIV transmission". (Levy, 1988). At least one other eminent HIV/AIDS researcher is also of the opinion that HIV cannot be transmitted through "...products prepared from blood, such as albumin, plasma, protein fractions, or hepatitis B vaccine" (Blattner, 1989). If HIV cannot be transmitted through "cell-free" body fluids (plasma) because it is not found in the plasma of 70% of seropositive individuals and in the remaining 30% is "generally at a concentration of less than 10 IP/mL", then it will be even less probable that factor VIII prepared from plasma can be a "meaningful source of HIV transmission" since, even if HIV were present in a plasma collection, it would be diluted many times over during the process of factor VIII manufacture. This is because factor VIII is made by pooling plasma obtained from 2000 to 30,000 individuals amongst whom at most, there will be only a few HIV seropositives. Since factor VIII prepared from large batches of pooled plasma is ultimately shared amongst many haemophiliacs, the load of HIV for each haemophiliac will be substantially lower than 10 IP/ml"
Basically, it seems that the typical Factor VIII collection and preperation methods of the '60s, '70s and early '80s may not have been capable of transmitting "HIV" or Hepatitis C as was widely reported.
http://www.sott.net/articles/show/197404-The-Flu-Epidemic-All-Fatal-Ukraine-Cases-at-GISAID-Have-RBD-D225G-Mutation
From Commenter Ruth.
"Recently saw a TV program about a geneticist who was studying human gene mutaion Delta 32. He discovered this interesting anomaly whilst studing a haemophiliac who had been given multiple injections of factor 8 for blood clotting (which at the time would have contained HIV). This haemophiliac never developed HIV because the gene mutation seemed to 'bounce' the virus off the cell receptor.
He linked its increased frequency in the population to the Plague which killed so many people in Europe 700 years ago. It's kind of odd that scientists are now querying whether Bubonic Plague was actually a virus.
[Link]
Its also odd that the highest frequency of this genetic mutation which is resistant to (some?)viruses should occur in the same areas that this Flu mutation now is (RBD D225G): Scandinavia and Russia. It all just seems a bit strange."
I would suggest reading this article "FACTOR VIII, HIV AND AIDS IN HAEMOPHILIACS: AN ANALYSIS OF THEIR RELATIONSHIP"
http://www.virusmyth.com/aids/hiv/ephemophilia.htm
posted section of the Abstract.
"Introduction
Currently, it is accepted that many patients with haemophilia have become HIV infected and/or developed the AID clinical syndrome as a direct result of the transfusion of factor VIII preparations contaminated with this particular virus. That this is indeed the case requires proof:
1. of the existence of a unique, infectious retrovirus, HIV. (For a critical discussion of this issue see Papadopulos-Eleopulos, 1988, Papadopulos- Eleopulos et al., 1992, Papadopulos-Eleopulos et al., 1993a, Papadopulos- Eleopulos et al., 1993b);
2. of the existence of HIV in factor VIII preparations;
3. of the existence of HIV in haemophiliacs;
4. that HIV is necessary and sufficient for the decrease in T4 cells observed in haemophiliacs;
5. that HIV and a decrease in T4 lymphocytes are necessary and sufficient for the development of the clinical AID syndrome.
Factor VIII and HIV
Since factor VIII is made from plasma, as a first step in proving contamination of this blood product with HIV, evidence must be presented that infectious viral particles with morphological characteristics attributed to HIV, are present in the plasma of "HIV infected" individuals. Then it must be shown that HIV can survive (a) the time between blood collection and freezing of plasma; (b) the freezing and thawing itself; © the process of manufacturing factor VIII from thawed plasma. In other words, as Jay Levy succinctly expressed in 1989, it is "important to know whether retroviruses could survive the preparation involved in producing Factor VIII concentrates. Otherwise, AIDS in many haemophiliacs [a minority may have other risk factors] could not be explained" (Levy, 1989).
HIV in plasma
To date, there is no evidence of the existence in human plasma of particles with the morphological characteristics attributed to HIV even though the plasma of at least some "HIV infected" individuals is claimed to contain such particles. Thus Levy, whose team reported most often on the relationship between HIV and factor VIII wrote in 1988: "Human Immunodeficiency virus in plasma or serum has been found in about 30% of specimens from seropositive persons, generally at a concentration of less than 10 IP/mL12" [IP=3Dinfectious particles] (Levy, 1988). Reference 12 cited by Levy is a paper which he published in collaboration with Barbara Michaelis but this paper does not contain a description of the method used to show that (a) HIV seropositive (non-haemophiliac) plasma was infected with "HIV particles"; (b) HIV was "present in low titers"; (c ) the particles were "infectious". Commenting on his and his colleagues' findings Levy wrote: "These studies demonstrate further that not all seropositive individuals have virus recoverable from their PMCs and that isolation from serum is not a common event" [PMCs=peripheral blood mononuclear cells] (Michaelis & Levy, 1987). "Thus, cell-free virus in body fluids is unlikely to be a meaningful source of HIV transmission". (Levy, 1988). At least one other eminent HIV/AIDS researcher is also of the opinion that HIV cannot be transmitted through "...products prepared from blood, such as albumin, plasma, protein fractions, or hepatitis B vaccine" (Blattner, 1989). If HIV cannot be transmitted through "cell-free" body fluids (plasma) because it is not found in the plasma of 70% of seropositive individuals and in the remaining 30% is "generally at a concentration of less than 10 IP/mL", then it will be even less probable that factor VIII prepared from plasma can be a "meaningful source of HIV transmission" since, even if HIV were present in a plasma collection, it would be diluted many times over during the process of factor VIII manufacture. This is because factor VIII is made by pooling plasma obtained from 2000 to 30,000 individuals amongst whom at most, there will be only a few HIV seropositives. Since factor VIII prepared from large batches of pooled plasma is ultimately shared amongst many haemophiliacs, the load of HIV for each haemophiliac will be substantially lower than 10 IP/ml"
Basically, it seems that the typical Factor VIII collection and preperation methods of the '60s, '70s and early '80s may not have been capable of transmitting "HIV" or Hepatitis C as was widely reported.
