Thanks to the group and especially to Laura and Andromeda for your constant hard work.
Don't know where to start with so many (of great bearing) question. The sessions have been selflessly provided for near 30 years, which is simply amazing, and each drills down further and further and connects and many align with our observable reality - and many don't. Of the 3D kind, this reality has simply gone haywire for at least the last decade, and of late, my God. Of the hyperdimensional aspects - chilling from a 3D perspective.
Sticking to the chemical kind of question:
Interesting, although by the numbers I don't know, that the U.S., Canada (Australia?) has a high statistic of what this all referenced. The EU must have a much lower statistic?
Of the G protein-coupled estrogen receptor:
And snip:
A whole lot more.
Don't know where to start with so many (of great bearing) question. The sessions have been selflessly provided for near 30 years, which is simply amazing, and each drills down further and further and connects and many align with our observable reality - and many don't. Of the 3D kind, this reality has simply gone haywire for at least the last decade, and of late, my God. Of the hyperdimensional aspects - chilling from a 3D perspective.
Sticking to the chemical kind of question:
Noted here:Q: (Joe) Does the presence of the weed killer atrazine in the food supply contribute to the increase in children and adolescents reporting as transgender?
wikiThere are over 300 products that contain atrazine {from Syngenta}.
Atrazine is in a group of man-made systemic herbicides called triazines {see here}.
Atrazine does not alter or damage genes in humans and animals.5,12,13,14 Female rats exposed to atrazine developed breast tumors. However, these tumors were not considered relevant to humans by a scientific advisory panel. This was because of differences in rat and human female reproductive systems.5 The EPA classified atrazine as "not likely to be carcinogenic to humans."1,5
A study in Texas found that mothers who lived in areas where more atrazine was used had a greater chance of giving birth to children with birth defects in their faces and skulls than mothers who did not. The more atrazine used in the county, the greater the chance of birth defects.21
It is used to prevent pre-emergence broadleaf weeds in crops such as maize (corn), soybean and sugarcane and on turf, such as golf courses and residential lawns. Atrazine's primary manufacturer is Syngenta and it is one of the most widely used herbicides in the United States, Canadian, and Australian agriculture. Its use was banned in the European Union in 2004, when the EU found groundwater levels exceeding the limits set by regulators, and Syngenta could not show that this could be prevented nor that these levels were safe.
Interesting, although by the numbers I don't know, that the U.S., Canada (Australia?) has a high statistic of what this all referenced. The EU must have a much lower statistic?
At least two significant Canadian farm well studies showed that atrazine was the most common contaminant found.[4] As of 2001, atrazine was the most commonly detected pesticide contaminating drinking water in the U.S.[8]: 44 Studies suggest it is an endocrine disruptor, an agent that can alter the natural hormonal system.[9]
Atrazine was invented in 1958 in the Geigy laboratories as the second of a series of 1,3,5-triazines.
Atrazine is prepared from cyanuric chloride, which is treated sequentially with ethylamine and isopropylamine. Like other triazine herbicides, atrazine functions by binding to the plastoquinone-binding protein in photosystem II, which animals lack. Plant death results from starvation and oxidative damage caused by breakdown in the electron transport process. Oxidative damage is accelerated at high light intensity.[26]
Atrazine's effects in humans and animals primarily involve the endocrine system. Studies suggest that atrazine is an endocrine disruptor that can cause hormone imbalance.[9]
Atrazine has been found to act as an agonist of the G protein-coupled estrogen receptor 1.[27] Atrazine has been shown to bind covalently to (chemically react with) a large number of mammalian proteins.[28]
Of the G protein-coupled estrogen receptor:
G protein-coupled estrogen receptor 1 (GPER), also known as G protein-coupled receptor 30 (GPR30), is a protein that in humans is encoded by the GPER gene.[5] GPER binds to and is activated by the female sex hormone estradiol and is responsible for some of the rapid effects that estradiol has on cells.[6]
Ligands[edit]
GPER binds estradiol with high affinity though not other endogenous estrogens, such as estrone or estriol, nor other endogenous steroids, including progesterone, testosterone, and cortisol.[6][9][10][11][12] Although potentially involved in signaling by aldosterone, GPER does not show any detectable binding towards aldosterone.[6][13][14] Niacin and nicotinamide bind to the receptor in vitro with very low affinity.[15][16] CCL18 has been identified as an endogenous antagonist of the GPER.[17] GPER-selective ligands (that do not bind the classical estrogen receptors) include the agonist G-1[18] and the antagonists G15[19] and G36.[20][6]
And snip:
Little information is available regarding the risks to children
The World Health Organization's International Agency for Research on Cancer has classified atrazine as "not classifiable as to its carcinogenicity to humans"
A whole lot more.