New Sally K. Norton interview by Mercola - Jan 1, 2023

I was dumping oxalate through my eyes, ears, scars, skin, urine, feces the first 6-8 months. I still do that but only occasionally, and then only in my urine. I've ben taking 10-20 g a day of ascorbate and still improving, but I will now test a new kind of Lipo-C (see Thors thread ) to see if I start feeling better/dumping, only 1-3 g a day.
How do you know you are dumping oxalates? I mean how can you tell?
 
Are y'all reducing your higher oxalate foods slowly or getting rid of them?
I'm currently wrestling with this a bit. I had cut out most plant foods earlier last year, but was still drinking lot's of tea which I was unaware is super high in oxalates. Not long after I switched my diet, I had all these hard white little bumps appear on the inner joints of my knees and elbows. Might be connected. They have yet to go away completely.
I've mostly had some bowel issues for several months now and I'm not sure if it's oxalate related or the Ioderal 50mg (Iodine) I started taking around October or heaven forbid, lactose intolerance to my liberal daily use of butter. I'm giving iodine a break for a bit to assess. I switched immediately from tea back to coffee, because that was accounting for way more than a normal daily oxalate intake. And I've been adding 1-2 servings of low oxalate plants per day to my carnivore regimen. Various winter squash, arugula/watercress, coconut yogurt. Will have to give it more time and see how it goes.
 
I still have some inulin (a prebiotic) sitting in my cupboard and looked up the oxalates content which comes from the chicory root. Apparently, it improves oxalate degrading activity in combination with probiotics. However, I don't know how helpful this, since inulin could contain oxalates:

Then I found the next bit about quercetin which "has an inhibitory effect on urinary crystal deposit formation".

Results:​


The viability of MDCK cells significantly decreased and malondialdehyde production increased in the presence of oxalate. However, co-exposure to quercetin inhibited the decrease in cell viability and inhibited the lipid peroxidation production induced by oxalate. In the animal study malondialdehyde production in group 3 significantly decreased compared to that in group 2. Catalase and superoxide dismutase activity was increased in group 3 compared to that in group 2. The number of crystals in kidneys in group 3 was decreased significantly compared to that in group 2.
See: American Urological Association

But then again, I don't know whether it is worth the bother if these supplements are chockfull of oxalates. Or only take them for a short while?
 
The lists are constantly being updated, and that one looks like it is from 2007. More recent ones can be found here:

http://www.lowoxalate.info/food_lists/cat_lod_food_chart.pdf
For people who are more visual:
I took the PDF file from that link, exported it to Excel, sorted and colored the data, and exported it back to PDF.

If there are more up-to-date lists you would like to share, I could re-work the data if it helps anyone. I personally find it easier to read with colors.
 

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I think the attack on vitamin C, quercetin, and other new information must be considered in light of this thread.
 
I think the attack on vitamin C, quercetin, and other new information must be considered in light of this thread.

Yeah, there seems to be a lot of ”to and fro” about vitamin C, for example. Keyhole wrote a great post about the issue earlier in this thread:

It personally think it ultimately comes down to context. I am not convinced that anything over 250mg vitamin C is converted into oxalate in everyone.

There are a few variables which can determine the rate of conversion into oxalate: 1. Concentration of free transition metals like copper and iron, 2. The redox environment ( are there a tonne of free radicals floating around which enhance the oxidation of ascorbate?) and 3. Someone's predisposition towards endogeous oxalate production.

Sally Norton/Susan Owens are very strict with vitamin C, and that is probably for good reason in their own personal situation. Likewise, I have personally seen TOO MANY people who get worse / their "oxalate symptoms" relapse and worsen when taking vitamin C to pass it off as coincidence. So it seems objectively true that their are a group of people who endogenously produce oxalate when they take vitamin C. This is supported by a body of literature also.

But.... is this the case of everyone, or even a lot of people? Probably not. High-dose vitamin C seems to help a lot of people, and even if it does convert to oxalate, those people appear to have a good handling capacity.

Therefore, like everything else, context is important. Both camps (the pro-vitamin C, and the anti-vitamin C) make valid points and seem to apply to different groups of people.
 
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