Vaccine-induced viral Reactivation!
mRNA vaccines significantly increase the likelihood of reactivating Herpes Zoster virus, a cohort study with over 2 million people…
“Conclusions: Consistent with the hypothesis, a higher incidence of HZ was statistically detectable post-COVID-19 vaccine. Accordingly, the eruption of HZ may be a rare adverse drug reaction to COVID-19 vaccines. Even though the molecular basis of VZV reactivation remains murky, temporary compromising of VZV-specific T-cell-mediated immunity may play a mechanistic role in post-vaccination pathogenesis of HZ. Note that VZV reactivation is a well-established phenomenon both with infections and with other vaccines (i.e. this adverse event is not COVID-19-specific).”
Here’s a meta-analysis confirming the same phenomenon, significantly increased likelihood of Herpes Zoster virus reactivation as well as many other herpes family virus reactivation after COVID-19 vaccination.
“The results of our proportion meta-analysis showed that the rate of VZV reactivation among those who received the COVID-19 vaccine was 14 persons per 1000 vaccinations (95% CI 2.97–32.80). Moreover, our meta-analysis for HSV reactivation showed the rate of 16 persons per 1000 vaccinations (95% CI 1.06–46.4). Furthermore, the evidence from case reports/series showed 149 cases of HHV reactivation.”
The reactivation of herpesviruses (HHV) in COVID-19 patients is evident in the literature. Several reports have been published regarding the reactivation of these viruses (HSV, VZV, EBV, and CMV) among those who got COVID-19 vaccines. In this study, we ...
ncbi.nlm.nih.gov
There is a wealth of research showing that herpes viruses have a causal role in many autoimmune and chronic inflammatory conditions…
“There is an increasing evidence linking infection with herpesviruses to the development of multiple autoimmune disorders. Large epidemiological studies suggested that susceptibility to multiple sclerosis (MS) is gained in early childhood, with viral infections acting as a trigger. Consequently, herpesviruses which are a childhood infections, are considered appropriate candidates contributing to MS development [64]. Herpesviruses also persist in the host as a latent infection, and when reactivated contribute to disease pathogenesis as observed in systemic autoimmune diseases (SADs) [65,66].”