AUTOIMMUNE DISEASES CAUSED BY AN INFECTION?

[Gaby]

After watching "Why can't I get better?" by Dr. Richard Horowitz (quoted earlier in this thread), I got his book. I hope it contains everything that he explains in his talks!!

I just watched this follow up video as well:

http://<iframe width="853" height="...=0" frameborder="0" allowfullscreen></iframe>

Here are some notes I took from the video:

-He describes the classical patient with co-infections which reminds me of the symptoms reported here as Herx reactions including the migrating pains, myalgias, malaise, febrile syndrome, stiff neck, brain fog, it is difficult to fall asleep, some days are better, etc. Some have leukopenia, trombocytopenia in their blood tests.
-He highlights the importance of mycoplasma infections, typically people who have Herx reactions with doxycycline and/or any other tetracycline.
-There are several species of borrelia discovered recently. So other than the classical borrelia burgdorferi (the only one tested on mainstream tests), there are new 100 species in the US and over 300 species worldwide. A specific species manifested as schizophrenia in one patient. She healed when treated for the infection.
- He has have European patients come to him and they test positive for a load of stuff that is typically not tested in Europe because it is thought that these diseases don't exist and/or are problematic in Europe despite being described in the medical literature.
- Just as reported here and in the SOTT page, infection by these "parasites" and viruses in general are on the rise, even 20% rise in 2014 if I heard correctly for some weird species. In short, it is increasing a lot in recent years. It is a sign of the time! "Plague Time!".
- He says that these are persistent infections, specially if not treated within the first 30 days in case of Lyme's disease and that he can prove it with his experience: some patients have had PCR positive tests even after years of taking antibiotics.
- Getting a tetracycline (i.e. doxy) ASAP is very important in case or Ehrlichiosis infection, just as it is with other infections such as Lyme's, Bartonella, ETC.
Bad news: the typical "gastroenteritis" syndrome typically thought to be caused by viruses could be a clinical manifestation of coinfections such as Ehrlichiosis, Tularemia, Rocky Mountain Fever, Lyme's disease, ETC.
- Heartland virus (we have stories on SOTT on that one) which is a newly discovered tick borne virus, resembles Ehlrlichia. It may cause fatigue, fever, diarrhea, etc. It does NOT respond to doxycycline. It is a virus.
- NO reliable blood test for emerging co-infections which might eclipse Lyme's disease within years as an epidemic. For example, borrelia miyamotoi, found in San Francisco, N.E., Canada. Presumably, the birds are carrying the ticks around the world. There are several borrelia species found in Europe, different from the typical one from Lyme's disease causing skin rashes and neurological problems.
- Lyme can be transmitted to the fetus in pregnancies. So other co-infections and women are not getting screened before they get pregnant.
- Babesiosis: increases severity of Lyme's disease. Manifested as fevers, chills, flushing, day or night sweats, unexplained cough and air hunger (shortness of breath), fatigue, joint aches, paresthesias, emotional lability, cognitive issues.
- Pscychiatric case with lots of emotional lability, depression, anxiety and sweats: think of babesia! Reminds me of patients who felt better after taking an antibiotic (i.e. azythromicin).
- He quotes a French patient who was treated for Lyme's disease for 5 years, he never got better until he was treated for Babesia. He says that doxy works pretty well in conjunction with an anti-malarial. It works amazingly well in babesia co-infections but it causes a lot of neuropsychiatric symptoms and dizziness (herx reactions?).
- He reminds people that some antibiotics raises the QT interval (an electrical parameter in the EKG, which can cause heart arrythmias or similar problems), i.e., cipro, macrolides! So careful with mixing drugs.
- In babesiosis, avoid cortisone because it reactivates latent bacteria.
- Lymes gives false positives for rheumatoid factor and ANA (a lupus autoantibody marker). So often, it is not an autoimmune disease, but a coinfection or Lyme's disease. He said at some point that Babesia can cause pleuritis, pericarditis, myocarditis, frequent in lupus.
- Most common co-infection after babesia, making people sick with mycoplasma: bartonella (cat scratch, trench fever, carrions disease from sand flies. But there are many more species not screened in tests. A nightmare for clinicians!!). Gives rash, enlarged lymph nodes. Atypical manifestations: encephalopathy, chronic dyemilinating neuropathy, vasculitis, radiculitis, eye problems (weird ones: retinal artery occlusion or bizarre stuff), arthritis, sarcoidosis, osteolytic lesions, etc. It gets treated with doxy and quinolones. If there are people very sick with neuropathies and they don't respond to multiple neuropathic drugs, think of lyme's and co-infection. God, I can think of quite a few people I've seen who are taking anti-epileptics, pain killers of all kinds including those 100 more potent than morphine, and still have nerve pain!
- Intracellular co-infections: mycoplasma, Q ever, bartonella, typhus, brucella... Two antibiotics work better than one in very sick people. I'm thinking that it is like our protocol: doxy, or doxy and cipro or a macrolide if too sick or no Herx reactions with doxy. I'm thinking that all the preparation before the protocol, i.e. heavy metal detox, diet, iron chelation, FIR sauna, really does help in this.
- Bartonella often gives horizontal rashes, like stretch marks which are "violet".
- Transplacental transmission: Bartonella, leptospirosis, borrelia, Lyme, Babesiosis, rocky mountain spotted fever.
- Mycoplasma fermetans (Gulf War Syndrome, what Garth Nicolson described/pioneered): exacerbates a lot of problems, standard tests don't pick it up, interacts with B lymphocytes causing autoimmune reactions and rheumatoid disease. It increases inflammatory cytokines. Explains the minocycline and plaquenil treatment success in Rheumatoid arthritis.
- Standard labwork doesn't work so when you give two main antibiotics, you treat a lot of stuff at the same time. Despite the many causes, it makes it very simple, use two main antibiotics.
- Migratory pain is a clue to Lyme's disease.
- Lyme's have biofilms.

I attached one of the slides which synthesis symptoms.

 

[Gaby]

Second week on the protocol. I did my metro days, and now I'm on doxy and alopurinol. I have a problem with alopurinol, the dose is too high for me. So I naturally adjusted the dose, that is, I'm throwing up after the third dose during the night or upon waking, and I also have diarrhea first thing in the morning. The allopurinol also causes a lot of brain fog and slowness in general, and it also gave me a temporary red eye. It makes the linings in my inner mouth sore.

I could understand how anyone would catch a secondary infection when your inner linings are sore. Secondary infections are described by Garth Nicolson in his papers. If it happens, he suggests amoxicilin with clavulanic (Augmentin). I'm taking vitamin C and it helps to heal the inner linings.

So I'll take less allopurinol, I'm after all, a small person.

I also have emotional lability, which started with the metro and continues with the allopurinol.

I'm looking forward to finish the allopurinol.

 

[Yas]

Thanks for the update Gaby! And lots of strength to you during the protocol!


I'd say I'm a small person too, so I suppose I should take a lower dose when I take it, is that right? My weight is 52 kilos. Is there a general guide we could follow regarding the Allopurinol dose / weight ratio? There is one for Metronidazole, but it's from 68 kilos up, what about smaller people?


I ask this in order to add the reference to the protocol guide.


On my side, I'm feeling much better now that I stopped the Nitazoxanide, my HS is also much better and I'm not feeling as much pain as I was feeling before. But I'm still quite tired. Tomorrow I have to take a higher dose of Nystatin, si I'll see how that goes.

 

[Gaby]

Thanks for the update Gaby! And lots of strength to you during the protocol!


I'd say I'm a small person too, so I suppose I should take a lower dose when I take it, is that right? My weight is 52 kilos. Is there a general guide we could follow regarding the Allopurinol dose / weight ratio? There is one for Metronidazole, but it's from 68 kilos up, what about smaller people?


I ask this in order to add the reference to the protocol guide.


On my side, I'm feeling much better now that I stopped the Nitazoxanide, my HS is also much better and I'm not feeling as much pain as I was feeling before. But I'm still quite tired. Tomorrow I have to take a higher dose of Nystatin, si I'll see how that goes.

Glad you're feeling better.

For the metro, we added the doses to some weight references. For my weight (56 kg, which is going down at this point), I calculated it with a rule of three. So I guess we'll have to do that. Mine is 2 after breakfast, 2 after lunch, one after dinner.

For the allopurinol it will have to be more intuitive. I'm cutting the last dose because it literally makes me feel sick to my stomach. I think that 300mgx2 for small people and 300mgx3 for bigger people is a good enough general outline. If vomit and diarrhea persists, then allopurinol 100mg will have to be chosen instead in order to reduce the dose, or you split the 300mg pills in order to reduce by 150mg the dose until vomit and diarrhea disappears.

There is a protocol to re-introduce alopurinol slowly but surely in cases of hyperuricemia. Pretty much you start with 100mg, then after two days 100mg x 3, then go up until maximum 300mg x3. But in this protocol, it is for anti-parasitical purposes. So it seems that higher (highest) doses are needed as an "induction phase".

My 2 cents!

 

[Gaby]

For data collection purposes, here are a couple of references which backs up what is explained in the video by Richard Horowitz "Co infections Presentation, Diagnosis and Treatment" available at _https://www.youtube.com/watch?v=O9a-2Nb2sbk and quoted yesterday in this thread.

Possible role of tick-borne infection in “cat-scratch disease”: Comment on the article by Giladi et al

_http://onlinelibrary.wiley.com/doi/10.1002/art.21925/full

To the Editor:

In the report of their excellent study of “cat-scratch disease”–associated arthropathy, Giladi et al provide a comprehensive review of chronic arthropathy caused by infection with Bartonella henselae (1). The authors state that this “often severe” arthropathy “should be classified in the infection-related arthropathy group, potentially similar to Lyme disease arthritides.” The similarity to Lyme disease may be more than coincidental.

Infection with B henselae has been recognized as an emerging tick-borne disease (2–9). The organism has been detected in questing Ixodes ticks in North America, Europe, and Asia (4–9), and in some areas the prevalence of B henselae in ticks is reportedly higher than the prevalence of Borrelia burgdorferi, the spirochetal agent of Lyme disease (4). Peromyscus leucopus, the white-footed mouse, serves as a reservoir for both B burgdorferi and B henselae (10). The fact that 72% of “cat-scratch disease” cases in the US occur in a seasonal pattern between June and December supports the notion of arthropod transmission of B henselae, and human infection via bites from flies, fleas, and mites may also occur (2, 9). Bartonella infection has been associated with sudden cardiac death in Swedish orienteers with extensive arthropod exposure (11).

Transmission of B henselae from ticks to humans has been postulated in conjunction with B burgdorferi. A recent study using a highly specific but relatively insensitive polymerase chain reaction assay showed that B henselae coinfection occurred in 22 of 86 patients (26%) with Lyme disease (2). As with other tick-borne coinfections such as babesiosis, ehrlichiosis, and anaplasmosis, coinfection with B henselae and B burgdorferi may lead to more severe symptoms of Lyme disease, including debilitating arthropathy and neuropsychiatric symptoms (2, 3, 12). Elimination of B henselae infection may be difficult in both animals and humans, and treatment of coinfection in chronic Lyme disease may require prolonged antibiotic therapy (12–14).

From a pathophysiologic standpoint, Bartonella species are unique among bacteria in their ability to induce angioproliferative lesions that result in cutaneous and enteric vasculopathy (15). Of interest, B henselae utilizes amino acid catabolism rather than glycolysis to derive energy from its host, generating ammonia in the process (16). Ammonia production may contribute to the encephalopathy reported in patients with chronic B henselae infection, and it may also play a role in arthropathy, as noted with other arthritogenic bacteria (17). It is unclear whether vasculopathy or ammoniagenesis is involved in the joint inflammation described by Giladi et al.

Because transmission of B henselae is not limited to cats, we propose that the term “Bartonella-associated arthropathy” should be used to identify this form of infection-related joint disease. The role of B henselae as an emerging tick-borne coinfection in chronic Lyme arthropathy merits further study.

Another one:

Rash Decisions about Southern Tick—Associated Rash Illness and Lyme Disease

_http://cid.oxfordjournals.org/content/42/2/306.long

Sir—Wormser et al. [1] discuss the “distinct clinical presentations” (p. 958) of Lyme disease in New York, compared with those of southern tick—associated rash illness (STARI) in Missouri. Unfortunately, it appears that little progress has been made in understanding the cause of STARI during the past decade [2]. Even worse, the accompanying editorial by Dennis [3] suggests that, in some circles, the understanding of Lyme disease ecology and epidemiology may have regressed during the same time period.

Wormser et al. [1] confirm that STARI is not caused by Borrelia burgdorferi, the agent of Lyme disease in the United States. The identical conclusion was drawn 10 years ago on the basis of microbiological analysis of STARI patients in Missouri [2]. The observation that these patients have a “milder” clinical illness [3, p. 962] when treated promptly with antibiotics may be misleading. Because patients with STARI were more likely to recall a tick bite and had a faster onset of rash, compared with patients with Lyme disease, it follows that patients with STARI were treated more promptly with antibiotics than the comparison group of Lyme disease patients was, and this difference could account for the more benign course of illness in the STARI patient group. The findings of Wormser et al. [1] also suggest that antibiotic therapy is effective against STARI, which supports an infectious etiology for the disease. Identification of the causative agent of this tickborne illness awaits more-assiduous microbiological and genetic analyses.

In the accompanying editorial, Dennis [3] tells us that “various rodents” (p. 966) are the principal reservoirs for B. burgdorferi, that the Lone Star tick, Amblyomma americanum, found in southern states, does not carry the spirochete that causes Lyme disease, and that “the preponderance of evidence indicates that Lyme disease occurs rarely, if at all, in states south of Maryland and Virginia” (p. 967). The implication of these statements is that tickborne diseases are limited by geography, and that an infectious etiology is less likely for a Lyme disease—like rash occurring in a region “where Lyme disease is not known to occur” [3, p. 966]. This viewpoint ignores the fact that B. burgdorferi has been detected in A. americanum ticks in Florida [4], that Lyme disease is diagnosed in Georgia at a rate 40 times higher than the national government surveillance rate [5], and that deer and migratory birds play a major role in the dissemination of ticks and tickborne diseases throughout the United States and other parts of the world [6–9]. Lyme disease is now found in every state except one, and the patchy distribution of infected ticks in certain areas, such as Northern California, points to a more random distribution of the disease than what was previously thought [10–12]. Thus, it is dangerous to argue that STARI is not related to Lyme disease on the basis of geography. It is more likely that we have not yet identified the causative infectious agent of STARI, and until we do, empirical antibiotic treatment would seem to be the safest course for patients with this Lyme disease—like illness. Given the scant progress that has been made in our understanding of STARI during the past 10 years, it is illogical to ask sick patients who respond to therapy with antimicrobial agents to avoid treatment until doctors better understand the disease.

 

[Gaby]

A recent fb update from Richard Horowitz which highlights the importance of a holistic approach to treat infections and co-infections:

Major universities are finally taking an interest in persister bacteria and their role in contributing to chronic symptoms in patients with Lyme disease. Dr Ying Zhang and researchers from Johns Hopkins University just published on Borrelia persisters in Emerging Microbes and Infections where they identified the gene expression profile for Bb persisters that survived antibiotic treatment with doxycycline and amoxicillin. They found differences in transporter genes, bacterial envelope protein coding genes, DNA repair related genes, bacterial chemotaxis genes, bacterial secretion genes, and genes encoding proteases. Comparison of the pathways of the doxycycline persisters and amoxicillin persisters revealed that they share several common features where some genes were up regulated and some down regulated. "These gene expression changes may play important roles in facilitating survival of B. burgdorferi persisters under antibiotic stress...and the upregulated genes identified in B. burgdorferi persisters may not only serve as targets for developing new drugs for more effective treatment but also antigens for developing diagnostic tests for persistent Lyme disease, and finally for developing therapeutic vaccines for improved treatment". The MSIDS map identifies up to 16 different reasons why patients may stay ill after classical treatment for Lyme disease. Persistent infection with borrelia species and co-infections certainly plays a large role in chronic illness, but we must also treat associated inflammation, autoimmune reactions, detoxify internal and external biotoxins, repair the damage caused by free radicals and oxidative stress which damage mitochondria, nerves, brain cells and internal organs, while balancing cytokines, hormones, and the microbiome. Once all of the associated factors on the 16 point MSIDS map have been adequately addressed, the vast majority of my patients improve. A large thanks goes out to the Global Lyme Alliance for their support of this research, and to Dr Zhang and his colleagues at Johns Hopkins for continuing to search for answers for this debilitating illness.

Accompanying paper:

Persister mechanisms in Borrelia burgdorferi: implications for improved intervention
_http://www.nature.com/emi/journal/v4/n8/full/emi201551a.html

Diet, heavy metal detox (EDTA and/or DMSA), FIR saunas, supplements, alternative remedies, stress relief, etc may well be the best preparation for the antibiotic protocol. Dealing with biofilms is also very important. In short, what we have discussed. This is a good reminder as to why.

 

[Gaby]

For data collection purposes, here are a couple of references which backs up what is explained in the video by Richard Horowitz "Co infections Presentation, Diagnosis and Treatment" available at _https://www.youtube.com/watch?v=O9a-2Nb2sbk and quoted yesterday in this thread.

I found and had a look to his slides. It is basically the same protocol, only that he always suggests the anti-malarials and recommends a low carb diet if there is no good response as far as I can see. The other way around sounds better! Specially when considering that anti-malarials are serious stuff.

Anyway, here are the links for the slides in case someone wants to check them out:

Treatment protocols for the chronically ill patients
_http://www.ilads.org/media/boston/slides/horowitz/horowitz.html

Classical and Integrative Medical Approaches in Chronic Lyme Disease: New Paradigms in Diagnosis and Treatment
_http://www.filariane.org/uploads/pathologies/protocole-horowitz.pdf

This last is actually the first youtube video posted in this thread by Richard Horowitz.

 

[canavare]

I found and had a look to his slides. It is basically the same protocol, only that he always suggests the anti-malarials and recommends a low carb diet if there is no good response as far as I can see. The other way around sounds better! Specially when considering that anti-malarials are serious stuff.

Anyway, here are the links for the slides in case someone wants to check them out:

Treatment protocols for the chronically ill patients
_http://www.ilads.org/media/boston/slides/horowitz/horowitz.html

Classical and Integrative Medical Approaches in Chronic Lyme Disease: New Paradigms in Diagnosis and Treatment
_http://www.filariane.org/uploads/pathologies/protocole-horowitz.pdf

This last is actually the first youtube video posted in this thread by Richard Horowitz.

Thank you very much for sharing Dr. Horowitz's Protocol.
Dr. Horowitz's Protocol seems to be a mix of the protocols of Dr. Steven Harris and Dr. Cowden.
Both healing strategy's and 11 more healing strategies are covered in this book : http://books.google.com.tr/books/about/Insights_Into_Lyme_Disease_Treatment.html?id=ZFMRKZ2uoW4C&hl=tr

FROM THE PUBLISHER: If you traveled the country for appointments with thirteen Lyme-literate health care practitioners, you would discover many of the cutting-edge therapies used to combat chronic Lyme disease. You would also spend thousands of dollars on hotels, plane tickets, and medical appointment fees-not to mention the time that it would take to embark on such a journey. Even if you had the time and money to travel, would the physicians have enough time to answer all of your questions? Would you even know which questions to ask? In this long-awaited book, health care journalist Connie Strasheim has done all the work for you. She conducted intensive interviews with thirteen of the world's most competent Lyme disease healers, asking them thoughtful, important questions, and then spent months compiling their information into organized, user-friendly chapters that contain the core principles upon which they base their medical treatment of chronic Lyme disease. The specific practitioners interviewed represent a variety of medical disciplines, including allopathic, naturopathic, complementary, chiropractic, homeopathic, and energy medicine. Two European physicians were also interviewed. PHYSICIANS INTERVIEWED: * Steven J. Harris, M.D., Redwood City, CA * Steven Bock, M.D., Rhinebeck, NY * Susan Marra, M.S., N.D., Seattle, WA * Ginger Savely, DNP, San Francisco, CA * W. Lee Cowden, M.D., M.D. (H), Panama City, Panama * Ingo D. E. Woitzel, M.D., Pforzheim, Germany * Ronald Whitmont, M.D., Rhinebeck, NY * Deborah Metzger, Ph.D., M.D., Los Altos, CA * Peter J. Muran, M.D., M.B.A., San Luis Obispo, CA * Nicola McFadzean, N.D., San Diego, CA * Marlene Kunold, HCP, Hamburg, Germany * Elizabeth Hesse-Sheehan, DC, CCN, Kirkland, WA * Jeffrey Morrison, M.D., New York, NY All aspects of treatment are covered, from anti-microbial remedies and immune system support, to hormonal restoration, detoxification, dietary and lifestyle choices. Furthermore, the book ponders patient and practitioner challenges of treating chronic Lyme disease, and offers helpful insights to the friends and families of those coping with chronic illness. Patients can use this book to get new treatment ideas and to educate their local physicians. Practitioners can use it to learn about and stay current on the latest therapies. Lyme disease treatment is complex and controversial, and this book puts the treatment information you need in the palm of your hand.

 

[Hesper]

Updated the Google doc with info from the thread, updated experiences on protocols, and added info from the book Plague Time.

https://docs.google.com/document/d/1uy2iTblKBKI162PkMe-lsdqUuLuiKBYYBMsebzfjk3Y/edit#

Wishing all of you on the Protocol a speedy recovery! Go get 'em.

 

[JEEP]

More on biofilms from Extreme Health Radio:

How Killing Biofilms Can Eliminate All Disease - Robert Von Sarbacher
https://www.youtube.com/watch?v=h_j60JBZlnY

The show covers other topics in addition to biofilms & if you want to get to the 'meat' of the issue, jump to 54:56 - pretty incredible info if legit. It does boil down to "the product of the century" called Methusula although the amazing properties of beets & other foods are also highlighted & promoted.

He elaborates on beets & the mini-beet protocol at this link:
https://www.youtube.com/watch?v=y1VmG1cv_Ec
In this presentation he states that even parasites & Lyme disease cannot survive beets because they are so pervasive.

 

[Laura]

More on biofilms from Extreme Health Radio:

How Killing Biofilms Can Eliminate All Disease - Robert Von Sarbacher
https://www.youtube.com/watch?v=h_j60JBZlnY

The show covers other topics in addition to biofilms & if you want to get to the 'meat' of the issue, jump to 54:56 - pretty incredible info if legit. It does boil down to "the product of the century" called Methusula although the amazing properties of beets & other foods are also highlighted & promoted.

He elaborates on beets & the mini-beet protocol at this link:
https://www.youtube.com/watch?v=y1VmG1cv_Ec
In this presentation he states that even parasites & Lyme disease cannot survive beets because they are so pervasive.

Jeep, can you quickly synopsize? I have VERY little time to watch videos...

 

[JEEP]

"Jeep, can you quickly synopsize? " - Laura

How Killing Biofilms Can Eliminate All Disease - Robert Von Sarbacher
https://www.youtube.com/watch?v=h_j60JBZlnY

Apparently, the website for extremehealthradio no longer exists (where Von Sarbacher's articles/research were posted) but they do have facebook page. There was no transcript of this particular show available.

Beginning of interview, Robert Von Sarbacher, Health Researcher, says he's been speaking 14 -17 hr a day about biofilms - "we found the product of the century that destroys biofilms on contact". The slow way to eliminate biofilms - eat turnips & apprx 2 wks will have eaten away all the biofilms from the intestinal tract > side effect of turnips/beets/parsnips/carrots are pure food for probiotics. Explains what biofilms are; eating massive amts of garlic will kill probiotics - only takes 1 germ to get thru (& w/ probiotics killed, germ unimpeded). Same for too much oregano oil that wasn't based in olive oil - kills too many probiotics. Talks about skin disorders that came into existence w/ antibiotics.

Cont.- antibiotics kills off intestinal flora but only 1 germ necessary to get thru & lay down biofilm which in essence Teflon coats the intestinal tract & no intestinal flora will ever grow there again. Antibiotics have side effects equivalent to candida (later attributes antibiotics responsible for candida- cites articles from 50s). Even w/ 10X amt of introduced probiotics for 10 yrs, cannot get them to repopulate - what goes in just goes out - must continuously introduce probiotics although symptoms gone. However, stopping probiotics then caused return of previous symtoms within a week. Univ of Pennsylvania in 2005 discovered the 'Teflon' inside intestinal tract called biofilm. Also found in bloodstream/kidneys/pancreas/brain.

Must have scrapings of intestinal tract to determine biofilm existence as they are invisible to camera used to inspect tract. Nobody tests for biofilms. If you didn't have biofilm, you'd have the best skin in the known universe. Intestinal flora produces B vitamins particularly Vit B5. Sulphur is food for probiotics (not obtainable from raw food). Beets are like pure stomach acid & will eat biofilms away over time. Those doing the mini-beet protocol w/ good stomach acid will not urinate red (due to beets) or even yellow - will be crystal clear, a sign of good digestion.

Biofilms are killed on contact w/ saponified minerals - one example of a saponified mineral is boron (water soluble); most are fat soluble such as yucca. Biosalts - basically Dawn dishwashing liquid manufactured by the body - digest 100% of fats in the body & not by stomach acid which is invisible to fats. Saliva is greatest on the planet - more chewing means less work for pancreas > chew food 30X before swallowing (nobody does it).

Beets are #1 for saponifying mineral content - basically stomach acid & biosalts combined in one. Will dissolve biofilm just not on contact & will dissolve just about everything else thus known to be the most detoxing substance known to human kind. Soon as it hits bloodstream starts destroying germs, viruses, bacteria, mold, mildew, yeast/candida - it eats it & dissolves it from the inside out because it's pure zinc sulphate. You don't need to digest your food if you have beets as they're equivalent to stomach acid. DO NOT combine (as in juicing) w/ burdock as you could dissolve yourself! The mini-beet protocol involves beet juice/asparagus juice followed by apples. Some people cannot use this protocol but no problem because of #1 biofilm killer known to mankind - Methusela! Way easier than eating beets & turnips. Ingested turnips heat up the intestinal tract & kill all the crap off of there but the intestinal flora love them & the intestines rebuild - better than cayenne pepper. Must cook vegetables correctly to preserve the food enzymes.

Methusela is a combination of saponified minerals, electrolyte minerals, yucca extract, estra extract, estra oil extract from fern/chocolate & something unnamed. Dr. Shaler, a biofilm expert, says once biofilms removed & even before you've had a chance to regrow all your intestinal flora, you'll have about 100Xs absorbtion of any herb you've previously been doing.

To acquire this, give Von Sarbacher a call - take 1/2 tsp twice a day in water or juice (says it tastes awful so recommends juice). Side effect is that it destroys bacteria. Probably a minumum use of two months along w/ taking probiotics to repopulate the intestinal flora. Could possibly work in two weeks, but hasn't been proven as of yet. It removes detox side effects like nothing else. Is a deep liver cleanse.

His phone: 940-233-0484

 

[SeekinTruth]

Thanks for the summary, JEEP. Might watch that video one of these early morning reading days. I've been taking about a 1/4 teaspoon of borax for boron a couple of weeks now. Seems to be doing something, as I have a bit more energy and don't need a nap if I didn't sleep enough (been averaging about 4.5 hours of sleep per night for several weeks). Also been taken bio-enhanced / stabilized R-Lipoic Acid again (after a long break) which is WAY more bio-available than regular Alpha Lipoic Acid (up to 4X maximum plasma concentration as standard ALA and 40X as non-stabilized R-LA). And another thing I started taking again (after another long break) is Inositol Hexaphosphate (IP-6), which I had gotten as an adjunct for the iron protocol (in addition to EDTA and decanting). It promotes proper calcium metabolism / crystalization processes in bones, also for renal, and vascular tissues. It can also inhibit oxidative reactions initiated by iron, one of the major contributors to the proliferation of free radicals. Finally IP-6 may support healthy regulation of cellular growth and reproduction, and enhanced activity of NK cells. So not sure which, if any or all of them, are having some positive results. FWIW.

 

[herondancer]

I did listen to the whole Von Sarbacher video. He throws around a lot of terms and skates off into made cow disease, canola oil and other stuff. Very disorganized and full of disinfo. I lived in an area where there was a big problem with mad cow. His assessments on that weren't even wrong. He couldn't even give a coherent answer as to what a rapeseed (canola) plant is. Ditto on dentistry. A lot of sweeping black and white statements ("Excema was unknown until vaccines." [???!]). it was sprinkled with some decent info, but it was stuff any good googler could find. It took over half the interview to get to biofilms, and then it boiled down to a pitch for his new product and his website. Interviewers were pretty clueless too. Not worth the time, imo. There's way better information out there. He did at least refute the raw food thing.'

If you've got the time, there is some good info there, but you'll have a lot of weeding to do.

Added: Von Sarbacher may very well have a somewhat useful protocol with the beet thing, I just really found his presentation very off-putting, full of grandiose claims. It was hard to get though the show.

 

[Laura]

Well, I love beets and turnips so that would be no hardship.