Re: Ketogenic Diet - Path To Transformation?
That may be so. However also it is good to recall the types of ketones being produced, some are not detected by the ketostix as we learned last summer, and there is this quote from "The Art and Science of Low Carbohydrate Living":
Gawan said:Breton said:So... in just a quick question, just checking if I get what is basically going on here:
do I understand from this that when we all (or many of us) went into ketosis around last summer, we probably have not stayed in ketosis all the time since then? This was often because (for many of us) we let our protein intake get to high and the conversion to glucose stopped or slowed it? So now this Intermittent Fasting and such is necessary to heal mtDNA and ALSO is a good way to get back into ketosis as well?
I can only write a short note on my experience, I did recently some ketone tests again (with a blood meter) and it showed something with 1.2, 2.0 and 5 at different times on the day. So it can be said I was in ketosis more or less. The intermittent fasting could help out further more getting into full ketosis. But I don't the definition what is full ketosis and what not. So it is something to further improve the state of ketosis imo (with new knowledge), or kind of a kick-starter.
That may be so. However also it is good to recall the types of ketones being produced, some are not detected by the ketostix as we learned last summer, and there is this quote from "The Art and Science of Low Carbohydrate Living":
Within a few days of starting on carbohydrate restriction, most people begin excreting ketones in their urine. This occurs before serum ketones have risen to their stable adapted level because un-adapted renal tubules actively secrete beta-hydroxybutyrate and acetoacetate into the urine. This is the same pathway that clears other organic acids like uric acid, vitamin C, and penicillin from the serum.
Meanwhile, the body is undergoing a complex set of adaptations in ketone metabolism[99]. Beta-hydroxybutyrate and acetoacetate are made in the liver in about equal proportions, and both are initially promptly oxidized by muscle. But over a matter of weeks, the muscles stop using these ketones for fuel. Instead, muscle cells take up acetoacetate, reduce it to beta-hydroxybutyrate, and return it back into the circulation. Thus after a few weeks, the predominant form in the circulation is beta-hydroxybutyrate, which also happens to be the ketone preferred by brain cells (as an aside, the strips that test for ketones in the urine detect the presence of acetoacetate, not beta-hydroxybutyrate).